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Ching-Ho Wang

Researcher at National Taiwan University

Publications -  69
Citations -  1384

Ching-Ho Wang is an academic researcher from National Taiwan University. The author has contributed to research in topics: Virus & Influenza A virus. The author has an hindex of 22, co-authored 68 publications receiving 1261 citations. Previous affiliations of Ching-Ho Wang include Council of Agriculture.

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Galectin-1 Binds to Influenza Virus and Ameliorates Influenza Virus Pathogenesis

Mei Lin Yang, +13 more
- 01 Oct 2011 - 
TL;DR: It is shown for the first time that intranasal treatment of galectin-1 could enhance survival of mice against lethal challenge with influenza virus by reducing viral load, inflammation, and apoptosis in the lung.
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Differential localization and function of PB1-F2 derived from different strains of influenza A virus

Chi-Jene Chen, +5 more
- 01 Oct 2010 - 
TL;DR: Although the swine-origin influenza virus (S-OIV) contained a truncated form of PB1-F2 (12 amino acids [aa), potential mutation in the future may enable it to contain a full-length product, therefore, the functions of this putative S-O IV PB1 -F2 were also investigated.
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Synergistic antiviral effect of Galanthus nivalis agglutinin and nelfinavir against feline coronavirus

Li-En Hsieh, +8 more
- 01 Oct 2010 - 
TL;DR: The results suggest that the combined use of GNA and nelfinavir has therapeutic potential in the prophylaxis and treatment of cats with early-diagnosed FIP.
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Rapid and specific influenza virus detection by functionalized magnetic nanoparticles and mass spectrometry

Tzu-Chi Chou, +8 more
- 16 Nov 2011 - 
TL;DR: The assay combining efficient magnetic separation and MALDI-MS readout offers a rapid and sensitive method for virus screening and a timely diagnosis within one hour.
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Inhibitory and combinatorial effect of diphyllin, a v-ATPase blocker, on influenza viruses.

Hui-Wen Chen, +9 more
- 01 Sep 2013 - 
TL;DR: Results show that diphyllin alters cellular susceptibility to influenza viruses through the inhibition of endosomal acidification, thus interfering with downstream virus replication, including that of known drug-resistant strains.