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Chitose Orikasa

Researcher at Nippon Medical School

Publications -  18
Citations -  778

Chitose Orikasa is an academic researcher from Nippon Medical School. The author has contributed to research in topics: Preoptic area & Estrogen receptor. The author has an hindex of 11, co-authored 17 publications receiving 740 citations.

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Distribution and hormone regulation of estrogen receptor immunoreactive cells in the hippocampus of male and female rats

TL;DR: Estrogen regulates the synaptic plasticity and physiology of the hippocampus as well as learning behaviors that are mediated by the hippocampus and can have a direct effect on hippocampal neurons and that any sex differences in estrogen responsiveness is due to something other than sex Differences in ER levels or function in the hippocampus.
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Sexually dimorphic expression of estrogen receptor β in the anteroventral periventricular nucleus of the rat preoptic area: Implication in luteinizing hormone surge

TL;DR: Findings provide support for the hypothesis that activation of ERβ in the AVPV is an important regulatory event in the female-typical induction of luteinizing hormone surge by estrogen.
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Estrogen configures sexual dimorphism in the preoptic area of C57BL/6J and ddN strains of mice

TL;DR: The calbindin‐immunoreactive cell aggregates in the preoptic area of C57BL/6J and ddN mice are homologous to the sexually dimorphic nucleus of the rat pre optic area in terms of the morphology and sex steroid‐dependent organization.
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Estrogen receptor alpha, but not beta, is expressed in the interneurons of the hippocampus in prepubertal rats: an in situ hybridization study

TL;DR: Non-isotopic, digoxigenin (dig)-labeled, in situ hybridization histochemistry for the both ER forms were applied and the distribution of their messages in serial, frontal sections over the postnatal period and in the adult.
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Exogenous estrogen acts differently on production of estrogen receptor in the preoptic area and the mediobasal hypothalamic nuclei in the newborn rat

TL;DR: Results of quantification of ER mRNA by reverse transcription-polymerase chain reaction correlated well with results from ISHH: that is, ER mRNA expression decreased in the MBH but not in the POA, indicating that estrogen affects ER gene expression differently in these two brain regions.