C
Chongshan Dai
Researcher at China Agricultural University
Publications - 66
Citations - 1751
Chongshan Dai is an academic researcher from China Agricultural University. The author has contributed to research in topics: Apoptosis & Oxidative stress. The author has an hindex of 20, co-authored 50 publications receiving 1081 citations. Previous affiliations of Chongshan Dai include Northeast Agricultural University & University of Texas Southwestern Medical Center.
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Colistin-Induced Nephrotoxicity in Mice Involves the Mitochondrial, Death Receptor, and Endoplasmic Reticulum Pathways
TL;DR: This is the first study to demonstrate that all three major apoptosis pathways and autophagy are involved in colistin-induced nephrotoxicity.
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Transcription factors in ferroptotic cell death.
TL;DR: Recent progress in understanding the transcriptional regulation underlying ferroptotic cell death is summarized, and how it has provided new insights into cancer therapy is discussed.
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Chloroquine ameliorates carbon tetrachloride-induced acute liver injury in mice via the concomitant inhibition of inflammation and induction of apoptosis
TL;DR: It is demonstrated that CQ ameliorates CCl4-induced acute liver injury via the inhibition of HMGB1-mediated inflammatory responses and the stimulation of pro-apoptotic pathways to modulate the apoptotic and inflammatory responses associated with progress of liver damage.
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Lycopene Attenuates Colistin-Induced Nephrotoxicity in Mice via Activation of the Nrf2/HO-1 Pathway
TL;DR: It is demonstrated that coadministration of 20 mg/kg/day lycopene can protect against colistin-induced nephrotoxicity in mice and its ability to activate the Nrf2/HO-1 pathway.
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Curcumin Attenuates on Carbon Tetrachloride-Induced Acute Liver Injury in Mice via Modulation of the Nrf2/HO-1 and TGF-β1/Smad3 Pathway
TL;DR: Curcumin could protect against CCl4-induced acute liver injury by inhibiting oxidative stress and inflammation, which may partly involve the activation of Nrf2/HO-1 and inhibition of TGF-β1/Smad3 pathways.