C
Christine Courteix
Researcher at University of Auvergne
Publications - 47
Citations - 2261
Christine Courteix is an academic researcher from University of Auvergne. The author has contributed to research in topics: Neuropathic pain & Hyperalgesia. The author has an hindex of 18, co-authored 45 publications receiving 2104 citations. Previous affiliations of Christine Courteix include French Institute of Health and Medical Research.
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Journal ArticleDOI
Streptozocin-induced diabetic rats: behavioural evidence for a model of chronic pain
TL;DR: This model is of interest as the observed reactions to noxious and non‐noxious stimuli correspond to hyperalgesia and allodynia, symptoms encountered in painful diabetic neuropathy in man.
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Oxaliplatin‐induced cold hypersensitivity is due to remodelling of ion channel expression in nociceptors
Juliette Descoeur,Vanessa Pereira,Vanessa Pereira,Anne Pizzoccaro,Anne Pizzoccaro,Amaury François,Amaury François,Bing Ling,Bing Ling,Violette Maffre,Violette Maffre,Brigitte Couette,Brigitte Couette,Jérôme Busserolles,Jérôme Busserolles,Christine Courteix,Christine Courteix,Jacques Noël,Michel Lazdunski,Alain Eschalier,Alain Eschalier,Nicolas Authier,Nicolas Authier,Emmanuel Bourinet,Emmanuel Bourinet +24 more
TL;DR: It is reported that oxaliplatin exaggerates cold perception in mice as well as in patients, and ivabradine may represent a tailored treatment for oxali platin‐induced neuropathy.
Journal Article
Antidepressants and pain
TL;DR: It is suggested that antidepressants have a genuine analgesic effect and that research into their mechanisms of action will help to facilitate the development of new drugs.
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Diabetes-induced mechanical hyperalgesia involves spinal mitogen-activated protein kinase activation in neurons and microglia via N-methyl-D-aspartate-dependent mechanisms.
Laurence Daulhac,Christophe Mallet,Christine Courteix,Monique Etienne,Eliane Duroux,Anne-Marie Privat,Alain Eschalier,Joseph Fialip +7 more
TL;DR: It is demonstrated for the first time that NMDA receptor-dependent phosphorylation of MAPKs in spinal cord neurons and microglia contribute to the establishment and longterm maintenance of painful diabetic hyperalgesia and that these kinases represent potential targets for pain therapy.
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Study of the sensitivity of the diabetes-induced pain model in rats to a range of analgesics.
TL;DR: The results demonstrate the high pharmacological predictivity of this model of painful diabetes and suggest that in this pathological condition, among the drugs acting on monoaminergic transmission, noradrenergic drugs seem the most active.