C
Christopher A. Jolly
Researcher at Texas A&M University
Publications - 15
Citations - 1096
Christopher A. Jolly is an academic researcher from Texas A&M University. The author has contributed to research in topics: Fatty acid & Fatty acid-binding protein. The author has an hindex of 15, co-authored 15 publications receiving 1069 citations.
Papers
More filters
Journal ArticleDOI
Dietary (n-3) Polyunsaturated Fatty Acids Suppress Murine Lymphoproliferation, Interleukin-2 Secretion, and the Formation of Diacylglycerol and Ceramide
TL;DR: Low dose, short term dietary exposure to highly purified EPA or DHA appears to suppress mitogen-induced T-lymphocyte proliferation by inhibiting IL-2 secretion, and these events are accompanied by reductions in the production of essential lipid second messengers, DAG and ceramide.
Journal ArticleDOI
Dietary Protein or Arginine Deficiency Impairs Constitutive and Inducible Nitric Oxide Synthesis by Young Rats
TL;DR: Because NO is a mediator of the immune response and is the endothelium-dependent relaxing factor, impaired NO synthesis may help explain immunodeficiency and cardiovascular dysfunction in protein- or arginine-deficient subjects.
Journal ArticleDOI
Fatty acid binding protein isoforms: structure and function
TL;DR: Which FABPs form biochemically defined or true isoforms versus FABP that form additional forms, operationally defined as isoforms, is critically evaluated.
Journal ArticleDOI
Fatty Acid Binding Protein: Stimulation of Microsomal Phosphatidic Acid Formation
TL;DR: Results demonstrate for the first time that both L-F ABP and I-FABP stimulate liver microsomal phosphatidic acid formation by enhancing synthesis of phosphatidate from fatty acyl-CoA and glycerol 3-phosphate.
Journal ArticleDOI
Effects of dietary n-3 fatty acids on T cell activation and T cell receptor-mediated signaling in a murine model.
TL;DR: Dietary EPA and DHA suppressed antigen-specific delayed hypersensitivity reactions and mitogen-induced proliferation of T cells and blunted the production of intracellular second messengers, including diacylglycerol and ceramide, following mitogen stimulation in vitro.