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Daniel G. Streicker

Researcher at University of Glasgow

Publications -  96
Citations -  5075

Daniel G. Streicker is an academic researcher from University of Glasgow. The author has contributed to research in topics: Desmodus rotundus & Rabies virus. The author has an hindex of 35, co-authored 85 publications receiving 3632 citations. Previous affiliations of Daniel G. Streicker include EcoHealth Alliance & University of Georgia.

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Host Phylogeny Constrains Cross-Species Emergence and Establishment of Rabies Virus in Bats

TL;DR: Using a data set of hundreds of rabies viruses sampled from 23 North American bat species, a general framework is presented to quantify per capita rates of cross-species transmission and reconstruct historical patterns of viral establishment in new host species using molecular sequence data, which demonstrate diminishing frequencies of both cross- species transmission and host shifts with increasing phylogenetic distance between bat species.
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Anthropogenic land use change and infectious diseases: a review of the evidence.

TL;DR: A systematic review 305 scientific articles investigating how specific types of anthropogenic land use change influence infectious disease dynamics, emphasizing the importance of ecological and evolutionary theory to understand pathogen responses in changing landscapes.
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Linking anthropogenic resources to wildlife-pathogen dynamics: a review and meta-analysis.

TL;DR: It is shown that food provisioning results in highly heterogeneous infection outcomes that depend on pathogen type and anthropogenic food source, and empirical support for behavioural and immune mechanisms through which human-provided resources alter host exposure and tolerance to pathogens is found.
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Multiple mortality events in bats: a global review.

TL;DR: Trends in the occurrence and apparent causes of multiple mortality events (MMEs) in bats around the world are reviewed and qualitatively described.
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Viral zoonotic risk is homogenous among taxonomic orders of mammalian and avian reservoir hosts

TL;DR: It is shown that the proportion of viruses that infect humans varies minimally across reservoir taxonomic orders, which supports a host-neutral explanation for observed variation in the number of zoonoses among animal groups, such that traits of animal orders are unlikely to produce viruses that disproportionately threaten humans.