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Daniel Navajas

Researcher at University of Barcelona

Publications -  289
Citations -  40206

Daniel Navajas is an academic researcher from University of Barcelona. The author has contributed to research in topics: Continuous positive airway pressure & Decellularization. The author has an hindex of 62, co-authored 275 publications receiving 35537 citations. Previous affiliations of Daniel Navajas include Autonomous University of Madrid & McGill University.

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Fibroblast viability and phenotypic changes within glycated stiffened three-dimensional collagen matrices

Vanesa Vicens-Zygmunt, +13 more
- 01 Jul 2015 - 
TL;DR: The use of non-enzymatic glycation with a low ribose concentration may provide a suitable model with a mechanic and oxidative modified environment with cells embedded in it, which allowed cell proliferation and induced fibroblast phenotypic changes.
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Obstructive apneas induce early release of mesenchymal stem cells into circulating blood.

Alba Carreras, +5 more
- 01 Jan 2009 - 
TL;DR: Application of a pattern of airway obstructions similar to those experienced by patients with sleep apnea induced an early mobilization of mesenchymal stem cells into circulating blood.
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Thrombin-induced contraction in alveolar epithelial cells probed by traction microscopy

Núria Gavara, +5 more
- 01 Aug 2006 - 
TL;DR: The contractile response of alveolar epithelial cells to the inflammatory agonist thrombin was mediated by actin cytoskeleton remodeling and actomyosin activation through myosin light chain kinase and Rho kinase signaling pathways.
Journal Article

In vitro comparative study of two decellularization protocols in search of an optimal myocardial scaffold for recellularization

Isaac Perea-Gil, +9 more
- 15 Mar 2015 - 
TL;DR: In this paper, decellularized myocardial scaffolds were reseeded by embedding them in RAD16-I peptidic hydrogel with adipose tissue-derived progenitor cells (ATDPCs).
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Inhomogeneity of local stiffness in the extracellular matrix scaffold of fibrotic mouse lungs.

Esther Melo, +6 more
- 01 Sep 2014 - 
TL;DR: Since acellular lungs from mice with bleomycin-induced fibrosis present considerable micromechanical inhomogeneity, this model can be a useful tool to better investigate how different degrees of extracellular matrix lesion modulate cell fate in the process of organ bioengineering from decellularized lungs.