D
Daniel Navot
Researcher at New York Medical College
Publications - 56
Citations - 2778
Daniel Navot is an academic researcher from New York Medical College. The author has contributed to research in topics: Ovulation & Follicular phase. The author has an hindex of 24, co-authored 56 publications receiving 2705 citations. Previous affiliations of Daniel Navot include Eastern Virginia Medical School & University of Palermo.
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Prognostic assessment of female fecundity
TL;DR: It is suggested that despite apparently normal ovulatory cycles, the DOR group has a compromised follicular apparatus and disparity between normal oestradiol secretory capacity of the granulosa and diminished capacity to secrete inhibin could explain the inappropriately high FSH levels in response to the CC challenge.
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Artificially Induced Endometrial Cycles and Establishment of Pregnancies in the Absence of Ovaries
Daniel Navot,Neri Laufer,Juri Kopolovic,Roni Rabinowitz,Arie Birkenfeld,Aby Lewin,Menachem Granat,Ehud J. Margalioth,Joseph G. Schenker +8 more
TL;DR: It is biologically feasible to simulate the essential hormonal and endometrial milieu of a fertile menstrual cycle and early gestation solely by the administration of estrogen and progesterone.
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The window of embryo transfer and the efficiency of human conception in vitro
Daniel Navot,Richard T. Scott,Kathleen Droesch,Lucinda L. Veeck,Hung-Ching Liu,Zev Rosenwaks +5 more
TL;DR: The results indicate a very high efficiency for in vitro fecundity provided optimal conditions are attained and the concepts leading to success in the ovum donation model should set the course for continued research toward improving results in other forms of assisted reproduction.
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Direct correlation between plasma renin activity and severity of the ovarian hyperstimulation syndrome.
Daniel Navot,Ehud J. Margalioth,Neri Laufer,Arie Birkenfeld,Abraham Relou,Ariel Rösler,Joseph G. Schenker +6 more
TL;DR: The present observation of high PRA in patients with OHSS may imply that the locally active renin angiotensin system, through induction of new vessel formation and increase in capillary permeability, may have a casual relationship to the ovarian enlargement and extracellular fluid accumulation that are the hallmarks of OHSS.
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Hormonal manipulation of endometrial maturation.
TL;DR: It is concluded that a very short exposure of the human endometrium to E or, conversely, prolonged E stimulation will allow normal endometrial maturation with the addition of P4, and Supraphysiological doses of P 4 in the accelerated secretory transformation protocol significantly enhanced endometrian maturational processes.