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Dario Caliandro

Researcher at Catholic University of the Sacred Heart

Publications -  12
Citations -  661

Dario Caliandro is an academic researcher from Catholic University of the Sacred Heart. The author has contributed to research in topics: Trophoblast & Antibody. The author has an hindex of 7, co-authored 8 publications receiving 637 citations. Previous affiliations of Dario Caliandro include The Catholic University of America.

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ANTIPHOSPHOLIPID ANTIBODIES AFFECT TROPHOBLAST GONADOTROPIN SECRETION AND INVASIVENESS BY BINDING DIRECTLY AND THROUGH ADHERED β2-GLYCOPROTEIN I

TL;DR: It is suggested that aPL recognition of both anionic PL and adhered beta2GPI on trophoblast cell structures might represent a potential pathogenetic mechanism for defective placentation in women with the antiphospholipid syndrome.
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Low-molecular weight heparin restores in-vitro trophoblast invasiveness and differentiation in presence of immunoglobulin G fractions obtained from patients with antiphospholipid syndrome

TL;DR: Low-molecular weight heparin, in a dose-dependent manner, significantly reduced the immunoglobulin G binding to trophoblast cells and restored in-vitro placental invasiveness and differentiation.
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Homocysteine Induces Trophoblast Cell Death with Apoptotic Features

TL;DR: It is suggested that trophoblast cell death might represent a pathogenic mechanism by which homocysteine may cause pregnancy complications related to placental diseases.
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Antiphospholid antibodies regulate the expression of trophoblast cell adhesion molecules

TL;DR: The results suggest that the inadequate trophoblastic invasion, induced by antiphospholipid antibodies, can be the result of abnormal trophoblast adhesion molecules expression.
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Monoclonal anti-annexin V antibody inhibits trophoblast gonadotropin secretion and induces syncytiotrophoblast apoptosis

TL;DR: It is suggested that recognition by anti-annexin V antibody of adhered annexin V on trophoblast cell structures might represent a potential pathogenic mechanism by which these antibodies can cause defective placentation.