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David Communi
Researcher at Université libre de Bruxelles
Publications - 74
Citations - 6442
David Communi is an academic researcher from Université libre de Bruxelles. The author has contributed to research in topics: Chemerin & Ligand (biochemistry). The author has an hindex of 25, co-authored 69 publications receiving 5988 citations. Previous affiliations of David Communi include Free University of Brussels.
Papers
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Journal ArticleDOI
vCSF Danger-associated Molecular Patterns After Traumatic and Nontraumatic Acute Brain Injury: A Prospective Study.
C. Santacruz,Jean Louis Vincent,Jorge Duitama,Edwin Bautista,Virginie Imbault,Michael Bruneau,Jacques Creteur,Serge Brimioulle,David Communi,Fabio Silvio Taccone +9 more
TL;DR: In this paper , the authors collected samples of ventricular cerebrospinal fluid (vCSF) for 5 days in 50 consecutive patients at risk of intracranial hypertension after traumatic and nontraumatic ABI.
Journal ArticleDOI
Proprioceptors-enriched neuronal cultures from induced pluripotent stem cells from Friedreich ataxia patients show altered transcriptomic and proteomic profiles, abnormal neurite extension, and impaired electrophysiological properties
Chiara Dionisi,Marine Chazalon,Myriam Rai,Céline Keime,Virginie Imbault,David Communi,Hélène Puccio,Serge N. Schiffmann,Massimo Pandolfo +8 more
TL;DR: In this article , the authors performed an in vitro characterization of human induced pluripotent stem cell-derived sensory neuronal cultures highly enriched for primary proprioceptive neurons, and the results indicated the existence of abnormalities affecting proprioceptors in Friedreich ataxia, particularly their ability to extend towards their targets and transmit proper synaptic signals.
Book ChapterDOI
Identification of Three Active Site Residues Involved in Substrate Binding by Human 43 kDa-D-myo-Inositol 1,4,5-Trisphosphate 5-Phosphatase
David Communi,Christophe Erneux +1 more
TL;DR: A specific Ins(1,3,4,5)P4-binding protein has been isolated and identified as a member of the GAP1 family, suggesting a connection between phospholipase C-derived signals and a proliferative cascade involving Ras.