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David G. Edwards

Researcher at University of Delaware

Publications -  121
Citations -  3301

David G. Edwards is an academic researcher from University of Delaware. The author has contributed to research in topics: Blood pressure & Kidney disease. The author has an hindex of 29, co-authored 105 publications receiving 2744 citations. Previous affiliations of David G. Edwards include College of Health Sciences, Bahrain & Christiana Care Health System.

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Dietary sodium and health: more than just blood pressure.

TL;DR: Pre-clinical and clinical data support that even in the absence of an increase in BP, excess dietary sodium can adversely affect target organs, including the blood vessels, heart, kidneys, and brain, and information and strategies for reducing dietary sodium are provided.
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Arterial elastance and wave reflection augmentation of systolic blood pressure: deleterious effects and implications for therapy.

TL;DR: Increased elastance (or stiffness, inverse of compliance) of the central elastic arteries is the primary cause of increased systolic and pulse pressure with advancing age and in patients with cardiovascular disease, including hypertension, and is due to degeneration and hyperplasia of the arterial wall.
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Exaggerated sympathetic and pressor responses to handgrip exercise in older hypertensive humans: role of the muscle metaboreflex.

TL;DR: Compared with NTN subjects, HTN adults exhibit exaggerated sympathetic and pressor responses to handgrip exercise that are maintained during PEI, indicating that activation of the metabolic component of the EPR is augmented in older HTN humans.
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Etiology of iliotibial band friction syndrome in distance runners.

TL;DR: Examination of differences between a noninjured cohort of runners and runners afflicted with iliotibial band friction syndrome according to selected anthropometric, biomechanical, muscular strength, and training measures revealed weekly mileage, and maximum normalized braking force to be the best discriminators.
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Effect of exercise training on endothelial function in men with coronary artery disease.

TL;DR: Twelve weeks of endurance exercise training led to an improvement in endothelial function as measured by brachial artery flow-mediated dilation and exercise training resulted in increased plasma nitrite and nitrate levels, increased plasma superoxide dismutase activity, and decreased oxidative stress.