scispace - formally typeset
S

Shannon L. Lennon

Researcher at University of Delaware

Publications -  43
Citations -  2483

Shannon L. Lennon is an academic researcher from University of Delaware. The author has contributed to research in topics: Internal medicine & Medicine. The author has an hindex of 16, co-authored 31 publications receiving 2275 citations. Previous affiliations of Shannon L. Lennon include University of Florida & Tel Aviv University.

Papers
More filters
Journal ArticleDOI

Mechanical Ventilation–induced Diaphragmatic Atrophy Is Associated with Oxidative Injury and Increased Proteolytic Activity

TL;DR: The hypothesis that MV is associated with atrophy of all diaphragmatic fiber types, increased diaphagmatic protease activity, and augmented diaphRAGmatic oxidative stress is supported.
Journal ArticleDOI

Analysis of cellular responses to free radicals: focus on exercise and skeletal muscle.

TL;DR: High-intensity exercise training has been shown to be generally superior to low- intensity exercise in the upregulation of muscle SOD and GPX activities and the effects of endurance training on non-enzymic antioxidants remain a relatively uninvestigated area.
Journal ArticleDOI

Resistance exercise and physical performance in adults aged 60 to 83.

TL;DR: This investigation examined the effect of 6 months of high‐ or low‐intensity resistance exercise on muscular strength and endurance and stair climbing ability in adults aged 60 to 83.
Journal ArticleDOI

Exercise, antioxidants, and HSP72: protection against myocardial ischemia/reperfusion.

TL;DR: It is concluded that exercise and antioxidants can independently provide protection against myocardial contractile dysfunction and infarction, and the combination of these two strategies does not enhance or inhibit the protection observed with each individual countermeasure.
Journal ArticleDOI

Short-term exercise training can improve myocardial tolerance to I/R without elevation in heat shock proteins

TL;DR: It is concluded that exercise-induced myocardial protection against a moderate duration I/R insult is not dependent on increases in myocardIAL HSPs, and it is postulate thatercise-associated cardioprotection may depend, in part, on rises in my cardiac antioxidant defenses.