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David J. Nott

Researcher at National University of Singapore

Publications -  171
Citations -  3735

David J. Nott is an academic researcher from National University of Singapore. The author has contributed to research in topics: Bayesian inference & Bayesian probability. The author has an hindex of 31, co-authored 157 publications receiving 3085 citations. Previous affiliations of David J. Nott include Clemson University & University of New South Wales.

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A comparative study of Markov chain Monte Carlo methods for conceptual rainfall‐runoff modeling

TL;DR: In this paper, Markov chain Monte Carlo (MCMCMC) sampling of the posterior distribution has been used to estimate parameter uncertainty in hydrological models, where prior knowledge about the parameter is combined with information from the available data to produce a probability distribution (the posterior distribution) that describes uncertainty about the parameters and serves as a basis for selecting appropriate values for use in modeling applications.
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Bayesian Synthetic Likelihood

TL;DR: The accuracy and computational efficiency of the Bayesian version of the synthetic likelihood (BSL) approach is explored in comparison to a competitor known as approximate Bayesian computation (ABC) and its sensitivity to its tuning parameters and assumptions.
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Adaptive sampling for Bayesian variable selection

David J. Nott, +1 more
- 01 Dec 2005 - 
TL;DR: In this article, an adaptive Monte Carlo sampling scheme for Bayesian variable selection in linear regression is proposed, which improves on standard Markov chain methods by considering Metropolis-Hastings proposals that make use of accumulated information about the posterior distribution obtained during sampling.
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Bayesian adaptive Lasso

TL;DR: In this paper, the authors proposed the Bayesian adaptive Lasso (BaLasso) for variable selection and coefficient estimation in linear regression, which is adaptive to the signal level by adopting different shrinkage for different coefficients.
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Meta-analysis and gene set enrichment relative to er status reveal elevated activity of MYC and E2F in the "basal" breast cancer subgroup.

TL;DR: Increased transcriptional activity of MYC is a characteristic of basal breast cancers where it mimics a large part of an estrogen response in the absence of the ER, suggesting a mechanism by which these cancers achieve estrogen-independence and providing a potential therapeutic target for this poor prognosis sub group of breast cancer.