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David K. Menon

Researcher at University of Cambridge

Publications -  822
Citations -  50694

David K. Menon is an academic researcher from University of Cambridge. The author has contributed to research in topics: Traumatic brain injury & Medicine. The author has an hindex of 102, co-authored 732 publications receiving 40046 citations. Previous affiliations of David K. Menon include Hammersmith Hospital & University of Leeds.

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Magnetic resonance for the anaesthetist. Part II : Anaesthesia and monitoring in MR units

TL;DR: In this article, a system for the management of critically ill patients and the conduct of anaesthesia in a magnetic resonance unit with a 1.6 tesla whole body magnet is described.
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Effect of cerebral perfusion pressure augmentation with dopamine and norepinephrine on global and focal brain oxygenation after traumatic brain injury.

TL;DR: If CPP is to be raised to a level higher than 65–70 mmHg, then it is important to recognise that the response to the intervention may be unpredictable and that the vasoactive agent used may be of importance.
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Proton MR spectroscopy of the brain in AIDS dementia complex

TL;DR: Spectra from patients with moderate to severe ADC exhibited significant reductions in levels of N-acetyl aspartate relative to creatine and also showed elevations in choline containing compounds (relative to Cr).
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Proton MR spectroscopy and imaging of the brain in AIDS: evidence of neuronal loss in regions that appear normal with imaging.

TL;DR: Proton magnetic resonance spectroscopy in two patients with AIDS is described and the role of 1H MRS in providing a marker of neuronal loss in patients with normal or borderline imaging is discussed.

of The Demographics and Clinical Assessment Working Group of the International and Interagency Initiative toward Common Data Elements for Research on Traumatic Brain Injury and Psychological Health

TL;DR: Criteria for considering or making a diagnosis of TBI is discussed, with a particular focus on the problems how a diagnosis can be made when patients present late after injury and how mild TBI may be differenti- ated from non-TBI causes with similar symptoms.