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David Zurakowski

Researcher at Boston Children's Hospital

Publications -  1230
Citations -  63131

David Zurakowski is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Medicine & Population. The author has an hindex of 117, co-authored 1168 publications receiving 55806 citations. Previous affiliations of David Zurakowski include Yale University & University of Pittsburgh.

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Cardiotoxicity associated with tyrosine kinase inhibitor sunitinib

TL;DR: Left ventricular dysfunction might be due, in part, to direct cardiomyocyte toxicity, exacerbated by hypertension, and patients treated with sunitinib should be closely monitored for hypertension and LVEF reduction, especially those with a history of coronary artery disease or cardiac risk factors.
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Functional recovery following traumatic spinal cord injury mediated by a unique polymer scaffold seeded with neural stem cells.

TL;DR: In this paper, the authors designed an implant modeled after the intact spinal cord consisting of a multicomponent polymer scaffold seeded with neural stem cells, and implanted it into an adult rat hemisection model of SCI.

Functional recovery following traumatic spinal cord injury mediated by a unique polymer scaffold seeded with neural stem cells

TL;DR: Implantation of the scaffold–neural stem cells unit into an adult rat hemisection model of SCI promoted long-term improvement in function relative to a lesion-control group, suggesting a possible regeneration component.
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Alignment in total knee arthroplasty. A comparison of computer-assisted surgery with the conventional technique.

TL;DR: Computer-assisted TKA gives a better correction of alignment of the leg and orientation of the components compared with the conventional technique, and potential benefits in the long-term outcome and functional improvement require further investigation.
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Elevated Glutamate Levels in the Vitreous Body of Humans and Monkeys With Glaucoma

TL;DR: The increased level of this known neurotoxin is consistent with an "excitotoxic" mechanism for the retinal ganglion cell and optic nerve damage in glaucoma and therapies to protect neurons against glutamate toxic effects may prove to be useful in the management of this blinding disease.