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Decui Shao

Researcher at Fudan University

Publications -  10
Citations -  231

Decui Shao is an academic researcher from Fudan University. The author has contributed to research in topics: Angiotensin II & Fibronectin. The author has an hindex of 8, co-authored 8 publications receiving 179 citations. Previous affiliations of Decui Shao include Fudan University Shanghai Medical College & Wannan Medical College.

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Journal ArticleDOI

H2S Inhibits Hyperglycemia-Induced Intrarenal Renin-Angiotensin System Activation via Attenuation of Reactive Oxygen Species Generation

TL;DR: Data suggested that the decrease in H2S under hyperglycemic condition leads to an imbalance between oxidative and reductive species, which, in turn, contributes to the pathogenesis of renal dysfunction.
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P300-dependent STAT3 acetylation is necessary for angiotensin II-induced pro-fibrotic responses in renal tubular epithelial cells

TL;DR: p300-dependent STAT3 acetylation is necessary for Ang II-induced STAT3 phosphorylation and the consequent pro-fibrotic responses in renal tubular epithelial cells in vitro.
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Suppression of XBP1S Mediates High Glucose-Induced Oxidative Stress and Extracellular Matrix Synthesis in Renal Mesangial Cell and Kidney of Diabetic Rats

TL;DR: The results suggested that XBP1S pathway of ER stress was involved in HG-induced oxidative stress and ECM synthesis, and a downstream target of X BP1S in regulating ROS formation might be NADPH oxidase.
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Inhibition of STAT3 acetylation is associated with angiotesin renal fibrosis in the obstructed kidney

TL;DR: STAT3 acetylation plays an important role in activation of STAT3 signaling pathway and consequent renal fibrosis in rats undergoing unilateral ureteral obstruction.
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Insulin deficiency induces rat renal mesangial cell dysfunction via activation of IGF-1/IGF-1R pathway.

TL;DR: Insulin deficiency increases the expression of IGF- 1 and IGF-1R in renal MCs and the kidney of diabetic rats, which contributes to the development of diabetic nephropathy.