D
Dwight W. Warren
Researcher at University of Southern California
Publications - 72
Citations - 2025
Dwight W. Warren is an academic researcher from University of Southern California. The author has contributed to research in topics: Lacrimal gland & Testosterone. The author has an hindex of 24, co-authored 72 publications receiving 1970 citations. Previous affiliations of Dwight W. Warren include LAC+USC Medical Center & Florida Atlantic University.
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Journal ArticleDOI
MHC class II molecules, cathepsins, and La/SSB proteins in lacrimal acinar cell endomembranes
Tao Yang,Hongtao Zeng,Jian Zhang,Curtis T. Okamoto,Dwight W. Warren,Richard L. Wood,Michael Bachmann,Austin K. Mircheff +7 more
TL;DR: Observations support the thesis that lacrimal gland acinar cells that have been induced to express MHC class II molecules function as autoantigen processing and presenting cells.
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Testosterone in the Fetal Rat Testis
TL;DR: Testes of fetal rats possess the enzymes necessary for converting radioactive acetate to radioactive testosterone as early as 15.5 days of intrauterine life, and the fetal testis is capable of formation do novo of testosterone through the period of sexual differentiation of the internal genitalia.
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Ontogeny of Gonadotropin Receptors in The Fetal and Neonatal Rat Testis
TL;DR: The data show that LH receptors are measurable at the time when the fetal testis becomes responsive to LH stimulation, and imply that this hormone may have a function in the developing gonad, particularly in the last 2 days of fetal life.
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Acquisition of Regulatory Mechanisms for Gonadotropin Receptors and Steroidogenesis in the Maturing Rat Testis
TL;DR: Testicular LH, lactogen, and estrogen receptors and in vitro steroidogenic and cAMP responses were measured in rats between the ages of 1 and 60 days, and there was no direct correlation between the concentrations of the two receptors.
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Effect of single dose dexamethasone on the concentration of serum triiodothyronine in man
TL;DR: The reduction in serum T3 in the athyreotic group on replacement thyroxine therapy demonstrates a direct effect of glucocorticoids on the peripheral metabolism of T3In man.