scispace - formally typeset
E

Edouard Akono Nantia

Researcher at University of Bamenda

Publications -  27
Citations -  511

Edouard Akono Nantia is an academic researcher from University of Bamenda. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 11, co-authored 23 publications receiving 410 citations. Previous affiliations of Edouard Akono Nantia include University of Yaoundé I.

Papers
More filters
Book ChapterDOI

Adverse Effects of Bisphenol A on Male Reproductive Function

TL;DR: The overall BPA effects on male reproduction appear to be more harmful if exposure occurs in utero, and the regulation of BPA and BPA-related products should be reinforced, particularly where exposure during the fetal period can occur.
Journal ArticleDOI

Medicinal plants as potential male anti-infertility agents: a review

TL;DR: Most of the data dealing with the effects of plant extracts on mammalian reproductive functions are summarized in this review.
Journal ArticleDOI

QuEChERS-based method for the determination of carbamate residues in aromatic herbs by UHPLC-MS/MS

TL;DR: A new reliable, fast and highly sensitive method based on ultra-high performance liquid chromatography tandem mass spectrometry has been developed and validated for the determination of 28 carbamates in aromatic herbs.
Journal ArticleDOI

Effect of agropesticides use on male reproductive function: a study on farmers in Djutitsa (Cameroon).

TL;DR: The results suggest that male farmers of Djutitsa (West Cameroon) are exposed to agropesticides due to improper protective tool, and this exposure may impair their reproductive function through inhibition of testosterone synthesis; probably by inhibition of testicular 17β‐ hydroxysteroid dehydrogenase (17HSD3) and induction of aromatase (CYP19).
Journal ArticleDOI

Effect of Environmental Contaminants on Mammalian Testis

TL;DR: It appears that environmental toxicants, especially heavy metals and organic chemicals of synthetic and microbiological origins, disrupt hormone production and action in the mammalian testes and thereby compromises the normal phenotypic development of male sexual characteristics, initiation and maintenance of spermatogenesis.