Elena Ladich

TL;DR: The Cypher and Taxus DES result in delayed arterial healing when compared with BMS of similar implant duration, and the cause of DES LST is multifactorial with delayed healing in combination with other clinical and procedural risk factors playing a role.

TL;DR: Neoatherosclerosis is a frequent finding in DES and occurs earlier than in BMS, and may be yet another rare contributing factor to late thrombotic events.

TL;DR: Vessel healing at the culprit site in AMI patients treated with DES is substantially delayed compared with the culprit sites in patients receiving DES for stable angina, emphasizing the importance of underlying plaque morphology in the arterial response to DES.

TL;DR: Compared with other causes of myocardial infarction, eosinophilic infiltrates are more common in thrombi harvested from very late DES thrombosis, particularly in sirolimus-eluting stents, and correlate with the extent of stent malapposition.

TL;DR: CoCr-EES demonstrated greater strut coverage with less inflammation, less fibrin deposition, and less late and very late stent thrombosis compared with SES and PES in human autopsy analysis, indicating that careful long-term follow-up remains important even after CoCr- EES placement.