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John B. Newell

Researcher at Harvard University

Publications -  116
Citations -  11424

John B. Newell is an academic researcher from Harvard University. The author has contributed to research in topics: Myocardial infarction & Mitral valve. The author has an hindex of 54, co-authored 116 publications receiving 11091 citations. Previous affiliations of John B. Newell include University of Connecticut & Genentech.

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Pathological Correlates of Late Drug-Eluting Stent Thrombosis Strut Coverage as a Marker of Endothelialization

TL;DR: Heterogeneity of healing is a common finding in drug-eluting stents with evidence of LST and demonstrates the importance of incomplete healing of the stented segment in the pathophysiology of L ST.
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Macrophage infiltration in acute coronary syndromes. Implications for plaque rupture.

TL;DR: Macrophage-rich areas are more frequently found in patients with unstable angina and non-Q-wave myocardial infarction, which suggests that macrophages are a marker of unstable atherosclerotic plaques and may play a significant role in the pathophysiology of acute coronary syndromes.
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Myocardial Phosphocreatine-to-ATP Ratio Is a Predictor of Mortality in Patients With Dilated Cardiomyopathy

TL;DR: The myocardial phosphocreatine-to-ATP ratio, measured noninvasively with 31P-MR spectroscopy, is a predictor of both total and cardiovascular mortality in patients with dilated cardiomyopathy.
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Pathology of Second-Generation Everolimus-Eluting Stents Versus First-Generation Sirolimus- and Paclitaxel-Eluting Stents in Humans

TL;DR: CoCr-EES demonstrated greater strut coverage with less inflammation, less fibrin deposition, and less late and very late stent thrombosis compared with SES and PES in human autopsy analysis, indicating that careful long-term follow-up remains important even after CoCr- EES placement.
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Prognostic value of exercise thallium-201 imaging in patients presenting for evaluation of chest pain

TL;DR: This study suggests an approach to evaluate the risk of future cardiac events in patients with possible ischemic heart disease by correlated with clinical, coronary and left ventricular angiographic and exercise electrocardiographic data over a 3 to 5 year period.