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Elijah R. Behr

Researcher at St George’s University Hospitals NHS Foundation Trust

Publications -  314
Citations -  14605

Elijah R. Behr is an academic researcher from St George’s University Hospitals NHS Foundation Trust. The author has contributed to research in topics: Brugada syndrome & Sudden cardiac death. The author has an hindex of 52, co-authored 274 publications receiving 11337 citations. Previous affiliations of Elijah R. Behr include University of London & St. George's University.

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Sudden cardiac death with autopsy findings of uncertain significance: potential for erroneous interpretation

TL;DR: In this article, the authors explored the hypothesis that sudden cardiac deaths represent sudden arrhythmic death syndrome (SADS) and found that a similar proportion of primary arrhythmogenic syndromes to a contemporary series of SADS.
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Drug-induced Brugada syndrome

TL;DR: The cases and evidence of drug-induced Brugada syndrome reported in the literature are reviewed to highlight the growing interest in the mechanisms responsible for this acquired ECG pattern and its clinical significance.
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The importance of specialist cardiac histopathological examination in the investigation of young sudden cardiac deaths

TL;DR: Expert cardiac pathology improves the accuracy of coronial post-mortem diagnoses in young SCD with fast-track reporting on hearts sent by pathologists in cases of SCD.
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Common variation in the NOS1AP gene is associated with drug-induced QT prolongation and ventricular arrhythmia.

TL;DR: It is shown that common variations in the NOS1AP gene may have relevance for future pharmacogenomic applications in clinical practice permitting safer prescription of drugs for vulnerable patients, and this results provide the first demonstration that common Variant NOS 1AP gene are associated with a significant increase in the risk of dLQTS.
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Calmodulin mutations and life-threatening cardiac arrhythmias: insights from the International Calmodulinopathy Registry.

TL;DR: Calmodulinopathies are largely characterized by adrenergically-induced life-threatening arrhythmias, and available therapies are disquietingly insufficient, especially in CALM-LQTS.