E
Enagnon Kazali Alidjinou
Researcher at university of lille
Publications - 100
Citations - 1171
Enagnon Kazali Alidjinou is an academic researcher from university of lille. The author has contributed to research in topics: Medicine & Coxsackievirus. The author has an hindex of 13, co-authored 78 publications receiving 658 citations. Previous affiliations of Enagnon Kazali Alidjinou include Lille University of Science and Technology.
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Hand, foot and mouth disease (HFMD): emerging epidemiology and the need for a vaccine strategy
TL;DR: The development of a globally representative multivalent HFMD vaccine could be the best strategy to protect against other major emerging etiologies of HFMD such as CV-A16,CV-A6 and CV- a10.
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Enteroviral pathogenesis of type 1 diabetes: queries and answers.
TL;DR: In this paper, a review focused on issues that are fueling debate, focusing on the interplay between genetic predisposition, immune system, and environmental factors, but a lot of controversies and unanswered questions remained, but it is still a challenge to date to prove a causal relationship between enterovirus and Type 1 diabetes.
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Preanalytical Issues and Cycle Threshold Values in SARS-CoV-2 Real-Time RT-PCR Testing: Should Test Results Include These?
Ilka Engelmann,Enagnon Kazali Alidjinou,Judith Ogiez,Quentin Pagneux,Sana Miloudi,Ilyes Benhalima,Mahdi Ouafi,Famara Sane,Didier Hober,Alain Roussel,Christian Cambillau,Christian Cambillau,David Devos,Rabah Boukherroub,Sabine Szunerits +14 more
TL;DR: In this paper, the authors provide some answers on how strongly preanalytical issues affect RT-PCR results by reviewing the utility of different transport buffer media and virus inactivation procedures and comparing the literature data with their own recent findings.
Journal Article
Enterovirus persistence as a mechanism in the pathogenesis of type 1 diabetes.
TL;DR: Enteroviral persistence is the result of a virus-host coevolution combining a cell resistance to lysis through mutations or down-regulation of viral receptor, and a decrease of the viral replication by genomic modifications or the production of a stable double-stranded RNA form.
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Impaired Functional T-Cell Response to SARS-CoV-2 After Two Doses of BNT162b2 mRNA Vaccine in Older People
Julie Demaret,Bénédicte Corroyer-Simovic,Enagnon Kazali Alidjinou,Anne Goffard,Jacques Trauet,Sophie Miczek,Fanny Vuotto,Arnaud Dendooven,Dominique Huvent-Grelle,Juliette Podvin,Daniel Dreuil,Karine Faure,Dominique Deplanque,Laurence Bocket,Alain Duhamel,Julien Labreuche,Annie Sobaszek,Michael Hisbergues,François Puisieux,Myriam Labalette,Guillaume Lefèvre +20 more
TL;DR: In this paper, the authors quantified S1-specific IgG, neutralizing antibody titers, total specific IFNγ secreting T cells, and functionality of CD4+ and CD8+ specific T cells by flow cytometry, after 2 doses of the BNT162b2 vaccine in younger and older people, with and without previous COVID-19 infection.