Erica J. Crespi

TL;DR: It is proposed that GC effects on life-history transitions, survival probabilities and fecundity can be modelled in existing quantitative demographic frameworks to improve the understanding of how GC variation influences life- history evolution and GC-mediated effects on population dynamics.

TL;DR: Findings support the concept that fetal growth retardation and postnatal catch-up growth, early markers of future adult diseases, can also be programmed by prenatal exposure to excess sex steroids.

TL;DR: It is shown that recombinant frog leptin (rxLeptin) is a potent anorexigen in frogs, as it is in mammals, but this response does not develop until midprometamorphosis, and there is evidence that leptin can influence limb growth and differentiation during early development.

TL;DR: Considerable evidence now supports the view that the secretion of hormones critical to development (corticosteroid and thyroid hormones) is controlled by a common neuroendocrine stress pathway involving corticotropin‐releasing factor (CRF) and related peptides.

TL;DR: There is an ontogenetic difference in the way the hypothalamic-pituitary-interrenal (HPI) axis responds to food deprivation in amphibians, and juvenile toads do not respond toFood deprivation by activating the HPI axis, but instead pursue a strategy of energy conservation that involves a reduction in plasma corticosterone concentration.