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Evelyne Dubois
Researcher at Université libre de Bruxelles
Publications - 92
Citations - 4743
Evelyne Dubois is an academic researcher from Université libre de Bruxelles. The author has contributed to research in topics: Saccharomyces cerevisiae & Gene. The author has an hindex of 39, co-authored 90 publications receiving 4596 citations. Previous affiliations of Evelyne Dubois include University of Tennessee Health Science Center.
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Premature termination of GAT1 transcription explains paradoxical negative correlation between nitrogen-responsive mRNA, but constitutive low-level protein production.
TL;DR: It is shown that the GAT1 gene, encoding a transcriptional activator of nitrogen-responsive catabolic genes, produces a variety of mRNAs differing in their 5′ and 3′ termini, which likely protects the cell from excess Gat1.
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Regulation of ureaamidolyase synthesis in Saccharomyces cerevisiae, RNA analysis, and cloning of the positive regulatory gene DURM.
TL;DR: The data suggest that the ureaamidolyase production is probably controlled at the level of transcription, and that two positive regulatory genes DURM and DURL are involved in the induction process.
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GATA Factor Regulation in Excess Nitrogen Occurs Independently of Gtr-Ego Complex-Dependent TorC1 Activation.
Jennifer J. Tate,Isabelle Georis,Rajendra Rai,Fabienne Vierendeels,Evelyne Dubois,Terrance G. Cooper +5 more
TL;DR: The data demonstrate the importance of searching for new mechanisms associated with TorC1 activation and/or the regulation of Gln3 localization/function in response to changes in the cells’ nitrogen environment, and the idea that TorC 1 activation may not be as central to nitrogen-responsive intracellular Gln 3 localization as envisioned previously.
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Nitrogen catabolite repression-sensitive transcription as a readout of tor pathway regulation: The genetic background, reporter gene and GATA factor assayed determine the outcomes (Genetics 181, (861-874))
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Constitutive and Nitrogen Catabolite Repression-Sensitive Production of Gat1 Isoforms
TL;DR: This is the first reported instance of Gln3 UASGATA-dependent protein production failing to derepress in nitrogen poor conditions, and regulated Gat1 production is more complex than previously recognized.