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Fabien Metivier

Researcher at French Institute of Health and Medical Research

Publications -  41
Citations -  7814

Fabien Metivier is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Blood pressure & End stage renal disease. The author has an hindex of 27, co-authored 41 publications receiving 7453 citations. Previous affiliations of Fabien Metivier include John Hunter Hospital.

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Arterial media calcification in end-stage renal disease: impact on all-cause and cardiovascular mortality

TL;DR: AMC is a strong prognostic marker of all-cause and CV mortality in HD patients, independently of classical atherogenic factors and the principal effect of AMC on arterial function is increased arterial stiffness.
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Arterial stiffening and vascular calcifications in end‐stage renal disease

TL;DR: The presence of vascular calcifications in ESRD patients was associated with increased stiffness of large capacity, elastic-type arteries, like the aorta and CCA, and the extent of arterial calcifications increased with the use of calcium-based phosphate-binders.
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Arterial Calcifications and Bone Histomorphometry in End-Stage Renal Disease

TL;DR: It is suggested that therapeutic interventions associated with excessive lowering of parathyroid activity (parathyroidectomy, excessive calcium or aluminum load) favor lower bone turnover and adynamic bone disease, which could influence the development and progression of AC.
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Cardiac and arterial interactions in end-stage renal disease

TL;DR: The present study documents parallel cardiac and vascular adaptation in ESRD, and demonstrates the potential contribution of structural and functional large artery alterations to the pathogenesis of left ventricular hypertrophy and functional alterations.
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Alterations of Left Ventricular Hypertrophy in and Survival of Patients Receiving Hemodialysis: Follow-up of an Interventional Study

TL;DR: Results show that a partial LVH regression in patients with ESRD had a favorable and independent effect on patients' all-cause and CV survival.