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Farah Khalil

Researcher at University of South Florida

Publications -  40
Citations -  1242

Farah Khalil is an academic researcher from University of South Florida. The author has contributed to research in topics: Cancer & Lung cancer. The author has an hindex of 13, co-authored 37 publications receiving 943 citations. Previous affiliations of Farah Khalil include Moffitt Cancer Center.

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Unique Ectopic Lymph Node-Like Structures Present in Human Primary Colorectal Carcinoma Are Identified by Immune Gene Array Profiling

TL;DR: Beneficial, intratumoral immune cell priming is suggested and the possibility of immunotherapy intervention decisions based on molecular signatures that can identify the presence of tumor-localized, ectopic lymph node-like structures is raised.
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PD-L1 expression is increased in a subset of basal type breast cancer cells.

TL;DR: Basal type breast cancer (especially basal B) express greater levels of PD-L1 constitutively and with IFN γ, which may enhance eradication of aggressive breast cancer cells by the immune system.
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Pathologist-level interpretable whole-slide cancer diagnosis with deep learning

TL;DR: A novel pathology whole-slide diagnosis method, powered by artificial intelligence, to address the lack of interpretable diagnosis, which provides an innovative and reliable means for making diagnostic suggestions and can be deployed at low cost as next-generation, artificial intelligence-enhanced CAD technology for use in diagnostic pathology.
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Down-regulation of Bax-interacting factor-1 in colorectal adenocarcinoma.

TL;DR: Bax‐interacting factor‐1 is a member of the endophilin B family that plays a critical role in apoptosis, autophagy, and mitochondrial morphology and the connection of Bif‐1 to colorectal cancer remains to be evaluated.
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Antagonism of adenosine A2A receptor expressed by lung adenocarcinoma tumor cells and cancer associated fibroblasts inhibits their growth

TL;DR: It is found that a significant number of lung adenocarcinomas express adenosine A2A receptors, and these observations add to the rationale for testing adenosines A 2A receptor antagonists as anticancer therapeutics.