F
Farzad Kahrizi
Researcher at Islamic Azad University
Publications - 6
Citations - 123
Farzad Kahrizi is an academic researcher from Islamic Azad University. The author has contributed to research in topics: Developmental toxicity & Oxidative stress. The author has an hindex of 4, co-authored 6 publications receiving 87 citations. Previous affiliations of Farzad Kahrizi include Shahid Beheshti University of Medical Sciences and Health Services.
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Interaction between the protective effects of cannabidiol and palmitoylethanolamide in experimental model of multiple sclerosis in C57BL/6 mice
TL;DR: Cannabinoids, non-psychoactive CBs, attenuate neurobehavioral deficits, histological damage, and inflammatory cytokine expression in MOG-immunized animals, and there is an antagonistic interaction between CBD and PEA in protection against Mog-induced disease.
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Curcumin attenuates bevacizumab-induced toxicity via suppressing oxidative stress and preventing mitochondrial dysfunction in heart mitochondria
TL;DR: The results indicate that the cardiotoxic effects of BEV are associated with mitochondrial dysfunction and ROS formation, which finally ends in MMP collapse and mitochondrial swelling as the “point of no return” in the cascade of events leading to apoptosis.
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Repeated Administration of Mercury Intensifies Brain Damage in Multiple Sclerosis through Mitochondrial Dysfunction.
Farzad Kahrizi,Ahmad Salimi,Farshid Noorbakhsh,Mehrdad Faizi,Freshteh Mehri,Parvaneh Naserzadeh,Nima Naderi,Jalal Pourahmad +7 more
TL;DR: Results showed that repeated treatment of mercury following induction of EAE in mice significantly increased the neurobehavioral scores, as well as mitochondrial toxicity through ROS formation, mitochondrial swelling, collapse of MMP and cytochrome c release.
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Bevacizumab as a monoclonal antibody inhibits mitochondrial complex II in isolated rat heart mitochondria: ameliorative effect of ellagic acid.
TL;DR: A significant decrease in activity of complexes II after exposure with bevacizumab is demonstrated and the protective effects of EA is proved in alleviating BEV-mediated mitochondria toxicity.
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Exposure to 4-methylimidazole as a food pollutant induces neurobehavioral toxicity in mother and developmental impairments in the offspring
TL;DR: Investigation in mother and offspring in mice showed that 4-MEI induces abortion, decrease the weight and length of the fetus and pathological abnormalities, and it is hoped that these results will be helpful for awareness of humans which are exposed with 4- MEI.