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Farideh Talebi

Researcher at Tehran University of Medical Sciences

Publications -  13
Citations -  330

Farideh Talebi is an academic researcher from Tehran University of Medical Sciences. The author has contributed to research in topics: Experimental autoimmune encephalomyelitis & Neuroinflammation. The author has an hindex of 7, co-authored 11 publications receiving 231 citations. Previous affiliations of Farideh Talebi include Shahed University.

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MicroRNA-142 regulates inflammation and T cell differentiation in an animal model of multiple sclerosis

TL;DR: It is suggested that increased expression of miR-142 isoforms might be involved in the pathogenesis of autoimmune neuroinflammation by influencing T cell differentiation, and this effect could be mediated by interaction ofmiR- 142 isoforms with SOCS1 and TGFBR-1 transcripts.
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Interaction between the protective effects of cannabidiol and palmitoylethanolamide in experimental model of multiple sclerosis in C57BL/6 mice

TL;DR: Cannabinoids, non-psychoactive CBs, attenuate neurobehavioral deficits, histological damage, and inflammatory cytokine expression in MOG-immunized animals, and there is an antagonistic interaction between CBD and PEA in protection against Mog-induced disease.
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MicroRNA-181 Variants Regulate T Cell Phenotype in the Context of Autoimmune Neuroinflammation

TL;DR: A significant decrease in miR-181a and -b levels in brain white matter from MS patients as well as in spinal cords of EAE mice during the acute and chronic phases of disease is revealed, highlighting the anti-inflammatory actions of miR -181a -b in the context of autoimmune neuroinflammation.
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Malat1 long noncoding RNA regulates inflammation and leukocyte differentiation in experimental autoimmune encephalomyelitis.

TL;DR: Analysis of the contributions of the MALAT1 long noncoding RNA to autoimmune neuroinflammation in central nervous system tissues from patients with multiple sclerosis and mice with experimental autoimmune encephalomyelitis points to a potential anti-inflammatory effect for MALat1 in the context of autoimmune neuro inflammation.
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MicroRNA-92a Drives Th1 Responses in the Experimental Autoimmune Encephalomyelitis.

TL;DR: The results indicate that miR-92a might affect Th1 differentiation, likely due to downregulation of TSC1 and DUSP10, and point to a potential role for miR -92a in neuroinflammatory responses in EAE.