F
Fatma M. El-Demerdash
Researcher at Alexandria University
Publications - 57
Citations - 3878
Fatma M. El-Demerdash is an academic researcher from Alexandria University. The author has contributed to research in topics: Lipid peroxidation & Antioxidant. The author has an hindex of 25, co-authored 42 publications receiving 3482 citations. Previous affiliations of Fatma M. El-Demerdash include Henan University.
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Cadmium-induced changes in lipid peroxidation, blood hematology, biochemical parameters and semen quality of male rats: protective role of vitamin E and β-carotene
TL;DR: Investigation of the potential protective effects of vitamin E and beta-carotene alone or in combination against cadmium (Cd) toxicity demonstrated the beneficial influences ofitamin E, -carotenes alone and/or in combination in reducing the harmful effects of CdCl2.
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Biochemical study on the hypoglycemic effects of onion and garlic in alloxan-induced diabetic rats
TL;DR: In this article, the effects of onion and garlic juices on biochemical parameters, enzyme activities and lipid peroxidation in alloxan-induced diabetic rats were investigated, and the results showed that garlic and onion juices exerted antioxidant and antihyperglycemic effects.
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Antioxidant effect of vitamin E and selenium on lipid peroxidation, enzyme activities and biochemical parameters in rats exposed to aluminium.
TL;DR: VE and Se have beneficial effects and could be able to antagonize Al toxicity, and VE or Se in combination with Al partially or totally alleviated its toxic effects on the studied parameters.
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Acrylamide-induced oxidative stress and biochemical perturbations in rats.
TL;DR: Different doses of acrylamide exerted deterioration effects on enzyme activities and lipid peroxidation in a dose-dependent manner and significantly reduced the content of sulfhydryl groups and protein in different tissues.
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Ameliorating effect of curcumin on sodium arsenite-induced oxidative damage and lipid peroxidation in different rat organs.
TL;DR: It can be concluded that curcumin has beneficial influences and could be able to antagonize Sa toxicity and reduce the induction in the levels of TBARS and induced the decrease in the activities of antioxidant enzymes and the Levels of SH-groups.