F
Fernando Peña
Researcher at CINVESTAV
Publications - 35
Citations - 2664
Fernando Peña is an academic researcher from CINVESTAV. The author has contributed to research in topics: Bursting & Glutamate receptor. The author has an hindex of 24, co-authored 35 publications receiving 2534 citations. Previous affiliations of Fernando Peña include National Autonomous University of Mexico & Instituto Politécnico Nacional.
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Journal ArticleDOI
Differential Contribution of Pacemaker Properties to the Generation of Respiratory Rhythms during Normoxia and Hypoxia
TL;DR: It is proposed that respiratory rhythm generation in normoxia depends on a heterogeneous population of pacemaker neurons, while during hypoxia the respiratory rhythm is driven by only one type ofpacemaker.
Journal ArticleDOI
Mecp2 deficiency disrupts norepinephrine and respiratory systems in mice.
Jean-Charles Viemari,Jean-Christophe Roux,Andrew K. Tryba,Véronique Saywell,Henri Burnet,Fernando Peña,Sebastien Zanella,Michelle Bévengut,Magali Barthelemy-Requin,Laura B K Herzing,Anne Moncla,Josette Mancini,Jan-Marino Ramirez,Laurent Villard,Gérard Hilaire +14 more
TL;DR: It is hypothesized that breathing disturbances in Mecp2-/y mice, and probably Rett patients, originate in part from a deficiency in noradrenergic and serotonergic modulation of the medullary respiratory network.
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Pacemaker neurons and neuronal networks: an integrative view
TL;DR: The nonlinearity of bursting activity might enable pacemaker neurons to facilitate the onset of excitatory states or to synchronize neuronal ensembles--an interactive process that is intimately regulated by synaptic and modulatory processes.
Journal ArticleDOI
Endogenous Activation of Serotonin-2A Receptors Is Required for Respiratory Rhythm Generation In Vitro
Fernando Peña,Jan-Marino Ramirez +1 more
TL;DR: It is concluded that endogenously activated 5-HT2A receptors are required for maintaining fictive respiratory activity in the brainstem slice by modulating sodium conductances via a PKC pathway.
Journal ArticleDOI
Seizures and neurodegeneration induced by 4-aminopyridine in rat hippocampus in vivo: role of glutamate- and GABA-mediated neurotransmission and of ion channels.
Fernando Peña,Ricardo Tapia +1 more
TL;DR: It is concluded that 4-aminopyridine stimulates the release of glutamate from nerve endings and that the resultant augmented extracellular glutamate is directly related to the neurodegeneration and is involved in the generation of epileptiform discharges through the concomitant overactivation of glutamate receptors.