scispace - formally typeset
F

Francesco Cipollone

Researcher at University of Chieti-Pescara

Publications -  228
Citations -  9635

Francesco Cipollone is an academic researcher from University of Chieti-Pescara. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 49, co-authored 195 publications receiving 8335 citations. Previous affiliations of Francesco Cipollone include University of Rome Tor Vergata & University of Messina.

Papers
More filters
Journal ArticleDOI

Cox-2 polymorphisms and cardiovascular disease: elucidating the hidden side of the disease.

TL;DR: It is now evident that conventional risk factors for myocardial infarcion, such as hypertension, diabetes, dyslipidemia and obesity all esult from the interaction between multiple susceptibility loci ith behaviour and the environment.
Journal ArticleDOI

Gastrointestinal manifestations of SARS-CoV-2 infection in an Italian population of hospitalized patients

TL;DR: This study on an Italian population during the outbreak of the COVID-19 pandemic shows that GI symptoms are part of the spectrum of the SARS-CoV-2 infection and could be the only manifestations at disease onset.
Journal ArticleDOI

Differential TBXA2 receptor transcript stability is dependent on the C924T polymorphism.

TL;DR: The instability of the TBXA2R transcript and the lack of effect on platelet aggregation might suggest a protective role for the TBXM2R TT genotype against atherothrombosis and its complications in high-risk aspirin-treated patients.
Journal ArticleDOI

Angiotensin II Blockade and Total Cardiovascular Risk

TL;DR: ACE inhibitors and ARBs are very important drugs in the modern management of the total cardiovascular risk in hypertensive patients because of the existence of blood pressure-independent effects.
Journal ArticleDOI

Flow-mediated vasodilation: improving specificity for endothelial function evaluation.

TL;DR: Under persistent pathological conditions such as hypertension, endothelial activity is greatly impaired, and endothelium undergoes rapid structural and functional changes that finally result in impaired vasorelaxation, oxidative stress and increased adhesiveness for circulating leukocytes (mainly monocytes and T-lymphocytes).