F
Francesco Cipollone
Researcher at University of Chieti-Pescara
Publications - 228
Citations - 9635
Francesco Cipollone is an academic researcher from University of Chieti-Pescara. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 49, co-authored 195 publications receiving 8335 citations. Previous affiliations of Francesco Cipollone include University of Rome Tor Vergata & University of Messina.
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Journal ArticleDOI
Determinants of enhanced thromboxane biosynthesis in patients with systemic lupus erythematosus
Domenico Ferro,Stefania Basili,S Roccaforte,M. Di Franco,Francesco Cipollone,Giovanni Ciabattoni,Giovanni Davì +6 more
TL;DR: A sensitive marker of platelet activation is characterized, which is abnormal in SLE patients who were positive for aPL and endothelial perturbation, which may help identify those patients at increased risk of thrombosis as potential candidates for antiplatelet therapy.
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Ultramicronized palmitoylethanolamide reduces viscerovisceral hyperalgesia in a rat model of endometriosis plus ureteral calculosis: role of mast cells.
Teresa Iuvone,Giannapia Affaitati,Daniele De Filippis,Mariangela Lopopolo,Gianluca Grassia,Domenico Lapenna,Luana Negro,Raffaele Costantini,Massimo Vaia,Francesco Cipollone,Armando Ialenti,Maria Adele Giamberardino +11 more
TL;DR: Ultramicronized palmitoylethanolamide significantly reduces VVH from ENDO+STONE, probably by modulating MC expression/activity in cysts, thus reducing central sensitization due to noxious signals from endometriotic lesions.
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Identification of microRNAs 758 and 33b as potential modulators of ABCA1 expression in human atherosclerotic plaques
Claudia Mandolini,Donato Santovito,P. Marcantonio,F. Buttitta,M. Bucci,Sante Ucchino,Andrea Mezzetti,Francesco Cipollone +7 more
TL;DR: Evidence of a strong posttranscriptional regulation of ABCA1 and ABCG1 expression in human atherosclerotic plaques from hypercholesterolemic patients is provided.
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Lp(a) and cardiovascular risk: Investigating the hidden side of the moon.
TL;DR: Lp(a) seems to increase CV risk through stimulation of platelet aggregation, inhibition of tissue factor pathway inhibitor, alteration of fibrin clot structure and promotion of endothelial dysfunction and phospholipid oxidation.
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Novel determinants of plaque instability
TL;DR: It is suggested that variable expression of upstream and downstream enzymes in the prostanoid biosynthetic cascade may represent important determinants of the functional consequences of COX‐2 expression and inhibition in different clinical settings.