F
Frida Berlin
Researcher at Lund University
Publications - 7
Citations - 27
Frida Berlin is an academic researcher from Lund University. The author has contributed to research in topics: Tryptase & Chymase. The author has an hindex of 1, co-authored 3 publications receiving 1 citations.
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Journal ArticleDOI
Mast Cell Proteases Tryptase and Chymase Induce Migratory and Morphological Alterations in Bronchial Epithelial Cells
TL;DR: In this paper, the effect of MC proteases on bronchial epithelial morphology and function was investigated, and the most significant increases were found in cell motility, cellular speed and cell elongation compared to non-stimulated cells.
Journal ArticleDOI
Mast cell tryptase enhances wound healing by promoting migration in human bronchial epithelial cells
Sofia Mogren,Frida Berlin,Sangeetha Ramu,Asger Sverrild,Celeste Porsbjerg,Lena Uller,Cecilia Andersson +6 more
TL;DR: In this paper, the role of tryptase and the protease-activated receptor-2 (PAR2) on epithelial wound healing is not fully investigated, however, the authors suggest that tryptases might be beneficial in tissue repair under baseline conditions.
Journal ArticleDOI
Direct effects of mast cell proteases, tryptase and chymase, on bronchial epithelial integrity proteins and anti-viral responses.
Sangeetha Ramu,Hamid Akbarshahi,Sofia Mogren,Frida Berlin,Samuel Cerps,Mandy Menzel,Morten Hvidtfeldt,Celeste Porsbjerg,Lena Uller,Cecilia Andersson +9 more
TL;DR: In this article, the authors investigated the effect of mast cells on the anti-viral response in bronchial epithelial cells (HBECs) during asthma exacerbations and found that mast cells play a critical role in asthma pathology by provoking pro-inflammatory and alarmin responses.
Journal ArticleDOI
Mast Cell Proteases Promote Diverse Effects on the Plasminogen Activation System and Wound Healing in A549 Alveolar Epithelial Cells
TL;DR: The data suggest a dual regulatory response from the MC proteases in events related to uPAR expression and wound healing which could be important features in asthmatic disease.