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G.D. Shanks

Researcher at United States Department of the Army

Publications -  11
Citations -  1036

G.D. Shanks is an academic researcher from United States Department of the Army. The author has contributed to research in topics: Malaria & Climate change. The author has an hindex of 9, co-authored 11 publications receiving 1003 citations. Previous affiliations of G.D. Shanks include Kenya Medical Research Institute.

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Climate change and the resurgence of malaria in the East African highlands.

TL;DR: Temperature, rainfall, vapour pressure and the number of months suitable for P. falciparum transmission have not changed significantly during the past century or during the period of reported malaria resurgence, suggesting claimed associations between local malaria resurgences and regional changes in climate are overly simplistic.
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Changing patterns of clinical malaria since 1965 among a tea estate population located in the Kenyan highlands.

TL;DR: The coincident arrival of chloroquine resistance during the late 1980s in the subregion suggests that drug resistance is a key factor in the current pattern and burden of malaria among this highland population.
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Successful double-blinded, randomized, placebo-controlled field trial of azithromycin and doxycycline as prophylaxis for malaria in western Kenya.

TL;DR: If studies with nonimmune volunteers confirm these results for semi-immune volunteers, a daily azithromycin regimen may have special utility for individuals with contraindications to treatment with doxycycline or other antimalarial agents.
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Efficacy and Safety of Atovaquone/Proguanil as Suppressive Prophylaxis for Plasmodium falciparum Malaria

TL;DR: Atovaquone/proguanil appears to be highly efficacious and safe as prophylaxis for P. falciparum malaria.
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Travel as a risk factor for uncomplicated Plasmodium falciparum malaria in the highlands of western Kenya.

TL;DR: Increased risk of malaria parasitaemia with travel was concentrated in children aged <5 years, and an increase in population gametocytaemia is possibly due to increased chloroquine resistance and suppressed infections contracted outside of the tea estates.