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G

G. Martinez-Escalera

Researcher at National Autonomous University of Mexico

Publications -  9
Citations -  103

G. Martinez-Escalera is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Lactation & Prolactin. The author has an hindex of 6, co-authored 9 publications receiving 102 citations.

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Release of catecholamines follows suckling or electrical stimulation of mammary nerve in lactating rats.

TL;DR: Results show that E and NE can be released in response to suckling, and that activation of ductal mechanoreceptors may inhibit such release, which may operate to regulate the rate of milk removal during suckling in the rat.
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A Solubility Shift Occurs during Depletion-Transformation of Prolactin within the Lactating Rat Pituitary*

TL;DR: The mechanism by which the depletion-transformation of PRL occurs within the anterior pituitary (AP) of the lacatating rat after suckling was investigated to determine the rate of loss in pH 10 extractable PRL from 0–60 min of suckling.
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Thiol Regulation of Depletion-Transformation and Release of Prolactin by the Pituitary of the Lactating Rat*

TL;DR: The concept that shifts in PRL detectability during depletion-transformation, repletion, and release involve thiol-disulfide interchange mechanisms is supported, whereas disulfides may inhibit depletion and facilitate PRL release.
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In-vivo and in-vitro secretion of prolactin by lactating rat adenohypophyses as a function of intracellular age

TL;DR: The [3H]prolactin concentration in the pituitary gland, analysed by polyacrylamide-gel electrophoresis, progressively fell as the time from labelling to removal of the pituitsary gland increased from 8 to 24h, which suggests that there was a loss of hormone as it aged within the gland.
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Reversal by thiols of dopamine-, stalk-median eminence-, and zinc-induced inhibition of prolactin transformation in adenohypophyses of lactating rats.

TL;DR: Thiol-disulfide interchange reactions may be importantly involved in both depletion-transformation and in secretion, and data indicate that thiols alone may inhibit rat PRL release and also facilitate or induce PRL depletion; in bovine PRL granules,Thiols reverse Zn++ inhibition of PRLrelease and detectability.