Maria Teresa Morales

TL;DR: Results show that E and NE can be released in response to suckling, and that activation of ductal mechanoreceptors may inhibit such release, which may operate to regulate the rate of milk removal during suckling in the rat.

TL;DR: It is shown that intact efferent sympathetic mammary innervation is required to restore both mammary D1 mRNA content and enzyme activity, whereas suckling-induced secretion of catecholamines from the adrenal glands does not seem to participate in M-D1 enzyme regulation.

TL;DR: It was found that biosynthesis and/or processing of PRL were markedly depressed in APs from rats whose pups were removed for 24 h, thus suggesting that a loss of labeled hormone occurred as it aged within the pituitary gland.

TL;DR: Thiol-disulfide interchange reactions may be importantly involved in both depletion-transformation and in secretion, and data indicate that thiols alone may inhibit rat PRL release and also facilitate or induce PRL depletion; in bovine PRL granules,Thiols reverse Zn++ inhibition of PRLrelease and detectability.

TL;DR: Results suggest that regulation of milk ejection may involve neurally mediated influences on mammary contractility and antagonistic alpha- and beta-adrenergic mechanisms may interact with each other to regulate milk ejections, and with afferent signals from the mammary glands.