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Geoffrey A. Head

Researcher at Baker IDI Heart and Diabetes Institute

Publications -  296
Citations -  10091

Geoffrey A. Head is an academic researcher from Baker IDI Heart and Diabetes Institute. The author has contributed to research in topics: Blood pressure & Baroreflex. The author has an hindex of 45, co-authored 278 publications receiving 8977 citations. Previous affiliations of Geoffrey A. Head include National Heart Foundation of Australia & Maastricht University.

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European Society of Hypertension Position Paper on Ambulatory Blood Pressure Monitoring

TL;DR: The historical background, the advantages and limitations of ABPM, the threshold levels for practice, and the cost-effectiveness of the technique are considered, while the role ofABPM in research circumstances, such as pharmacological trials and in the prediction of outcome in epidemiological studies is examined.
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European Society of Hypertension practice guidelines for ambulatory blood pressure monitoring.

TL;DR: The requirements and the methodological issues to be addressed for using ABPM in clinical practice are addressed, the clinical indications for ABPM suggested by the available studies are outlined in detail, and the place of home measurement of blood pressure in relation to ABPM is discussed.
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Vagal and sympathetic components of the heart rate range and gain of the baroreceptor-heart rate reflex in conscious rats

TL;DR: The results suggest that the baroreceptor-HR reflex in the conscious rat is best characterised by a sigmoid curve with approximately equal contributions from both the cardiac vagus and the sympathetic nerves.
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Determinants of cardiac fibrosis in experimental hypermineralocorticoid states

TL;DR: It is concluded that the myocardial fibrosis observed in response to chronic mineralocorticoid elevation and salt loading is a humorally mediated event independent of hypokalemia, hypertension, and cardiac hypertrophy.
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Genetic Ace2 Deficiency Accentuates Vascular Inflammation and Atherosclerosis in the ApoE Knockout Mouse

TL;DR: Genetic Ace2 deficiency is associated with upregulation of putative mediators of atherogenesis and enhances responsiveness to proinflammatory stimuli in atherosclerosis-prone ApoE KO mice, and these findings emphasize the potential utility of ACE2 repletion as a strategy to reduce Atherosclerosis.