George M. Vaughan

TL;DR: In a study of 36 men burned in a fire, based on sequential early morning samples, plasma cortisol concentration was elevated in proportion to burn size, suggesting that factors other than ACTH contribute to the elevated cortisol as mentioned in this paper.

TL;DR: Nonsurvival after burn injury was associated with reduced T4, FT4I, and mental status for up to weeks before death, this association being independent of treatment with drugs acting on mental status or thyroid function.

TL;DR: It is suggested that a reciprocal relationship exists between plasma concentration of T3 and both norepinephrine and epinephrine in untreated burn patients and that treatment with the metabolically active hormone, triiodothyronine, does not alter the level of hypermetabolism accompanying thermal injury.

TL;DR: Melatonin is not a marker for general sympathetic activity, even following severe burn injury, and appears partitioned, with that controlling melatonin (nocturnal surge) regulated independently.

TL;DR: Cutaneous thermal injury can cause resetting of the mechanism linking plasma tonicity and AVP secretion resulting in dilutional hyponatremia, which occurs in the absence of gross physiologic perturbations such as volume depletion or adrenal insufficiency.