G
Giovanni E. Salvi
Researcher at University of North Carolina at Chapel Hill
Publications - 10
Citations - 2111
Giovanni E. Salvi is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Periodontitis & Prostaglandin E2. The author has an hindex of 9, co-authored 10 publications receiving 1822 citations. Previous affiliations of Giovanni E. Salvi include University of Bern.
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Journal ArticleDOI
Potential Pathogenic Mechanisms of Periodontitis-Associated Pregnancy Complications
Steven Offenbacher,Heather Jared,P. G. O'Reilly,S. R. Wells,Giovanni E. Salvi,Herenia P. Lawrence,Sigmund S. Socransky,James D. Beck +7 more
TL;DR: New experiments from 48 case-control subjects have measured gingival crevicular fluid (GCF) levels of PGE(2) and IL-1-beta to determine whether mediator levels were related to current pregnancy outcome, and microbes associated with mature plaque and progressing periodontitis were detected at higher levels in PLBW mothers, as compared to NBW controls.
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Consensus report: Peri-implant diseases and conditions
Tord Berglundh,Gary C. Armitage,Gustavo Avila-Ortiz,Mauricio G. Araújo,Juan Blanco,Paulo M. Camargo,Stephen T. Chen,David L. Cochran,Jan Derks,Elena Figuero,Christoph H. F. Hämmerle,Lisa J. A. Heitz-Mayfield,Guy Huynh-Ba,Vincent J. Iacono,Ki-Tae Koo,Laurie K. McCauley,Marc Quirynen,Stefan Renvert,Giovanni E. Salvi,Frank Schwarz,Dennis P. Tarnow,Cristiano Tomasi,Hom-Lay Wang,Nicola U. Zitzmann +23 more
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Inflammatory mediator response as a potential risk marker for periodontal diseases in insulin-dependent diabetes mellitus patients.
Giovanni E. Salvi,Giovanni E. Salvi,Behnaz Yalda,J. G. Collins,Bettye H. Jones,Frances W. Smith,Roland R. Arnold,Steven Offenbacher +7 more
TL;DR: The data suggest that the high GCF and monocytic secretion of PGE2 and IL-1 beta in IDDM patients may be a consequence of a systemic response trait and that the presence of Gram-negative infections such as periodontal diseases may interact synergistically to yield high local levels of these mediators and a more severeperiodontal condition.
Journal ArticleDOI
Influence of risk factors on the pathogenesis of periodontitis
Journal ArticleDOI
Inflammatory mediators of the terminal dentition in adult and early onset periodontitis
Giovanni E. Salvi,Giovanni E. Salvi,C. E. Brown,Kohtaro Fujihashi,Hiroshi Kiyono,Frances W. Smith,James D. Beck,Steven Offenbacher +7 more
TL;DR: The data suggest that this cross-sectional TD periodontitis model may reflect progressive periodontal disease associated with tooth loss, and although Thl cytokine levels in the GCF dominate over the Th2 response, monocytic activation provides the main source of proinflammatory mediators.