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Giovanni Federspil

Researcher at University of Padua

Publications -  121
Citations -  4809

Giovanni Federspil is an academic researcher from University of Padua. The author has contributed to research in topics: Insulin & Insulin resistance. The author has an hindex of 33, co-authored 121 publications receiving 4592 citations. Previous affiliations of Giovanni Federspil include University of Geneva & University of Liège.

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Prevalence and Characteristics of the Metabolic Syndrome in Primary Aldosteronism

TL;DR: A negative effect of aldosterone excess on glucose metabolism is confirmed and the recently reported higher rates of cardiovascular events in primary aldosteronism than in essential hypertension might be due to increased prevalence of the metabolic syndrome in the former condition.
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Reduced Plasma Visfatin/Pre-B Cell Colony-Enhancing Factor in Obesity Is Not Related to Insulin Resistance in Humans

TL;DR: Visfatin is not related to insulin resistance either as assessed by homeostasis model assessment or during lipid infusion, whereas visfatin mRNA is differentially regulated in SAT and VAT.
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Plasma adiponectin is decreased in nonalcoholic fatty liver disease.

TL;DR: Data support a role for low circulating adiponectin in the pathogenesis of NAFLD and confirm the strict association between reduced adiponECTin production by adipose tissue, NAFLd and insulin resistance.
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The origin of intermuscular adipose tissue and its pathophysiological implications.

TL;DR: It is proved that muscle satellite cells (SCs), a heterogeneous stem cell population characterized by plasticity and self-renewal that allow muscular growth and regeneration, can acquire features of adipocytes, including the abilities to express adipocyte-specific genes and accumulate lipids.
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The endogenous cannabinoid system stimulates glucose uptake in human fat cells via phosphatidylinositol 3-kinase and calcium-dependent mechanisms.

TL;DR: A role for the local endocannabinoids in the regulation of glucose metabolism in human adipocytes is indicated and a role in channeling excess energy fuels to adipose tissue in obese humans is suggested.