G
Giulia De Lorenzo
Researcher at Sapienza University of Rome
Publications - 105
Citations - 8752
Giulia De Lorenzo is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Arabidopsis & Arabidopsis thaliana. The author has an hindex of 46, co-authored 101 publications receiving 7716 citations. Previous affiliations of Giulia De Lorenzo include University of Georgia & James I University.
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A domain swap approach reveals a role of the plant wall-associated kinase 1 (WAK1) as a receptor of oligogalacturonides
TL;DR: A chimeric receptor approach is adopted to elucidate the role of Arabidopsis WAK1 and shows that transgenic plants overexpressing Wak1 are more resistant to Botrytis cinerea and are effective against fungal and bacterial pathogens.
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Arabidopsis local resistance to Botrytis cinerea involves salicylic acid and camalexin and requires EDS4 and PAD2, but not SID2, EDS5 or PAD4.
TL;DR: Data indicate that local resistance to B. cinerea requires ethylene-, jasmonate-, and SA-mediated signaling, that the SA affecting this resistance does not require ICS1 and is likely synthesized via PAL, and that camalexin limits lesion development.
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Activation of defense response pathways by OGs and Flg22 elicitors in Arabidopsis seedlings
Carine Denoux,Roberta Galletti,Nicole Mammarella,Suresh Gopalan,Daniele Werck,Giulia De Lorenzo,Simone Ferrari,Frederick M. Ausubel,Julia Dewdney +8 more
TL;DR: Results suggest a lower threshold for activation of early responses than for sustained or SA-mediated late defenses, and expression patterns of amino-cyclopropane-carboxylate synthase genes also implicate ethylene biosynthesis in regulation of the late innate immune response.
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Oligogalacturonides: plant damage-associated molecular patterns and regulators of growth and development
Simone Ferrari,Daniel Valentin Savatin,Francesca Sicilia,Giovanna Gramegna,Felice Cervone,Giulia De Lorenzo +5 more
TL;DR: Oligogalacturonides are considered true damage-associated molecular patterns that activate the plant innate immunity and may also be involved in the activation of responses to mechanical wounding.
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Resistance to Botrytis cinerea Induced in Arabidopsis by Elicitors Is Independent of Salicylic Acid, Ethylene, or Jasmonate Signaling But Requires PHYTOALEXIN DEFICIENT3
Simone Ferrari,Roberta Galletti,Carine Denoux,Giulia De Lorenzo,Frederick M. Ausubel,Julia Dewdney +5 more
TL;DR: In Arabidopsis, OGs increase resistance to the necrotrophic fungal pathogen Botrytis cinerea independently of jasmonate (JA)-, salicylic acid (SA)-, and ethylene (ET)-mediated signaling.