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Gregory J. Raugi

Researcher at University of Washington

Publications -  58
Citations -  2247

Gregory J. Raugi is an academic researcher from University of Washington. The author has contributed to research in topics: Extracellular matrix & Teledermatology. The author has an hindex of 27, co-authored 58 publications receiving 2137 citations. Previous affiliations of Gregory J. Raugi include Washington University in St. Louis & United States Department of Veterans Affairs.

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Light microscopic immunolocation of thrombospondin in human tissues.

TL;DR: Results indicate that thrombospondin is located within the matrix of a variety of human tissues and supports the suggestion that this glycoprotein is an endogenous component of some extracellular matrices.
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Detection of Human Papillomavirus DNA in Cutaneous Squamous Cell Carcinoma among Immunocompetent Individuals

TL;DR: The high prevalence of HPV DNA detected among controls suggests that HPV DNA is widely distributed among the general population, but the differential detection of HPV beta-papillomavirus species in tumors among cases suggests that certain HPV types may be involved in the progression of cutaneous SCCs.
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Implications of the changing face of Clostridium difficile disease for health care practitioners

TL;DR: Faced with an increasing incidence of C difficile infections and the changing profile of patients who become infected, this paper will reexamine the current concepts on the epidemiology and treatment, present new hypotheses for risk factors, examine the role of spores in the transmission of C Difficile, and provide recommendations that may enhance infection control practices.
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Phenotypic expression of collagen types in mesangial matrix of diabetic and nondiabetic rats.

TL;DR: These studies demonstrate that glomerular hypertrophy and an increase in mesangial matrix occur in both hyperglycemic and insulin-treated rats; however, the mesangia matrix composition varies with the metabolic environment.
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Fluoroquinolone Use and Risk Factors for Clostridium difficile-Associated Disease within a Veterans Administration Health Care System

TL;DR: The increase in the number of cases of C. difficile-associated disease was not attributable to a formulary change of fluoroquinolones; instead, the incidence was within expected seasonal variations for C. diffusion disease.