Gregory P. Beehler

TL;DR: Evidence to date generally supports an association between breast cancer and polycyclic aromatic hydrocarbons and polychlorinated biphenyls in conjunction with certain genetic polymorphisms involved in carcinogen activation and steroid hormone metabolism.

TL;DR: There is no association that is consistent, strong, and statistically significant, with the exception of alcohol intake, overweight, and weight gain, among the prospective epidemiologic studies conducted on diet and breast cancer incidence and gene‐diet interactions.

TL;DR: Findings support a protective effect of coffee intake on premenopausal, but not postmenopausal breast cancer risk, and no clear associations between intake of black tea or decaffeinated coffee and Breast cancer risk were noted among premenographic women.

TL;DR: The chemoprotective effects of phytochemicals in black tea and decaffeinated coffee may be important, although the effects ofPhytochemicalicals in regular coffee may been counteracted by the elevated risk associated with its higher caffeine content.

TL;DR: A potential influence of menopausal status on the total endogenous hormonal environment, including estrogen, androgens, and insulin-like growth factors, when considering the association between body size and ovarian cancer risk is suggested.