H
Hai B. Dai
Researcher at Beth Israel Deaconess Medical Center
Publications - 22
Citations - 607
Hai B. Dai is an academic researcher from Beth Israel Deaconess Medical Center. The author has contributed to research in topics: Isoflurane & Vasodilation. The author has an hindex of 13, co-authored 22 publications receiving 593 citations.
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The Direct Vasomotor Effect of Thyroid Hormones on Rat Skeletal Muscle Resistance Arteries
TL;DR: It is concluded that thyroid hormones possess direct vasodilatory effects with both endothelium-independent and endothelia-dependent components.
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Microvascular endothelial dysfunction and its mechanism in a rat model of subarachnoid hemorrhage
TL;DR: After subarachnoid hemorrhage (SAH), large cerebral arteries are prone to vasospasm and cortical arterioles demonstrate endothelial dysfunction, which may be the basis for microvascular spasm, related to decreased NOS3, which occurs despite an increase in its transcription.
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Propofol-associated dilation of rat distal coronary arteries is mediated by multiple substances, including endothelium-derived nitric oxide
TL;DR: It is concluded that propofol has a direct vasodilatory effect on distal coronary arteries in rats and is primarily endothelium-dependent and is mediated by multiple substances, including nitric oxide (NO) and a vasodILatory prostanoid.
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Epithelial Dependence of the Bronchodilatory Effect of Sevoflurane and Desflurane in Rat Distal Bronchi
TL;DR: Bronchodilation by the volatile anesthetics desflurane and sevoflurane is at least partially epithelium-dependent and may be attenuated in diseases affecting the epithelia, such as asthma.
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Attenuation of endothelium-dependent dilation of pig pulmonary arterioles after cardiopulmonary bypass is prevented by monoclonal antibody to complement C5a.
Kyung W. Park,Motohisa Tofukuji,Caroline Metais,Mark E. Comunale,Hai B. Dai,Michael Simons,Gregory L. Stahl,Azin Agah,Frank W. Sellke +8 more
TL;DR: It is concluded that pig pulmonary endothelial dysfunction associated with CPB may be mediated by C5a, the mechanism may involve changes in NOS translation, and this decrease was prevented by a previous administration of MAb.