H
Han Cheng
Researcher at Anhui Medical University
Publications - 14
Citations - 411
Han Cheng is an academic researcher from Anhui Medical University. The author has contributed to research in topics: Umbilical vein & Angiogenesis. The author has an hindex of 8, co-authored 12 publications receiving 228 citations.
Papers
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Journal ArticleDOI
Association between air pollution and cardiovascular mortality in Hefei, China: A time-series analysis.
Chao Zhang,Rui Ding,Changchun Xiao,Yachun Xu,Han Cheng,Furong Zhu,Ruoqian Lei,Dongsheng Di,Qihong Zhao,Jiyu Cao +9 more
TL;DR: Findings showed that air pollution could significantly increase the CVD mortality, and women were more vulnerable than man upon air pollution exposure upon exposure to air pollution.
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The short-term effects of air pollution on respiratory diseases and lung cancer mortality in Hefei: A time-series analysis.
Furong Zhu,Rui Ding,Ruoqian Lei,Han Cheng,Jie Liu,Chaowei Shen,Chao Zhang,Yachun Xu,Changchun Xiao,Xiaoru Li,Junqing Zhang,Jiyu Cao +11 more
TL;DR: It is demonstrated that air pollution could evidently increase the respiratory disease and lung cancer mortality in Hefei, China, from 2009 to 2015.
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ROS-AKT-mTOR axis mediates autophagy of human umbilical vein endothelial cells induced by cooking oil fumes-derived fine particulate matters in vitro.
Rui Ding,Chao Zhang,Xiaoxia Zhu,Han Cheng,Furong Zhu,Yachun Xu,Ying Liu,Longping Wen,Jiyu Cao +8 more
TL;DR: Findings demonstrate ROS‐AKT‐mTOR axis plays a critical role in HUVECs autophagy induced by COFs‐derived PM2.5 via the PI3K/AKT/mTOR pathway.
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Effects of long-term exposure to air pollution on the incidence of type 2 diabetes mellitus: a meta-analysis of cohort studies.
TL;DR: The findings show long-term exposure to PM 2.5, and PM 10 could significantly increase the incidence of type 2 diabetes mellitus, especially in cohorts with long follow-up time, as well as educational level and gender.
Journal ArticleDOI
The effects of cooking oil fumes-derived PM2.5 on blood vessel formation through ROS-mediated NLRP3 inflammasome pathway in human umbilical vein endothelial cells.
Chaowei Shen,Jie Liu,Furong Zhu,Ruoqian Lei,Han Cheng,Chao Zhang,Xinmiao Sui,Liu Ding,Mei Yang,Hongbo Chen,Rui Ding,Jiyu Cao +11 more
TL;DR: It was revealed that the impact caused by COFs-derived PM2.5 on blood vessel formation through a ROS-mediated NLRP3 inflammasome pathway could significantly reduce HUVECs viability, induce the overproduction of ROS, lead to inflammation and inhibit VEGF expression, thus affect angiogenesis of HUVES in vitro.