H
Heather B Madsen
Researcher at Florey Institute of Neuroscience and Mental Health
Publications - 18
Citations - 481
Heather B Madsen is an academic researcher from Florey Institute of Neuroscience and Mental Health. The author has contributed to research in topics: Extinction (psychology) & Dopamine receptor D2. The author has an hindex of 12, co-authored 18 publications receiving 391 citations. Previous affiliations of Heather B Madsen include Monash University, Clayton campus & University of Melbourne.
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Drug versus sweet reward: greater attraction to and preference for sweet versus drug cues.
TL;DR: This study confirms that rats prefer the sweet lever because sweet water is more reinforcing and attractive than cocaine or heroin.
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Ontogeny of memory: An update on 40 years of work on infantile amnesia
TL;DR: Evidence is presented that infantile amnesia is unlikely to be explained by a unitary theory, with the protracted development of multiple brain regions and neurotransmitter systems important for learning and memory likely to be involved.
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Neuroplasticity in addiction: cellular and transcriptional perspectives
TL;DR: A summary of more recent research that has furthered understanding of drug-induced neuroplastic changes both at the level of the synapse, and on a transcriptional level, and how these changes may relate to the human disease of addiction is provided.
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Investigation of the neuroanatomical substrates of reward seeking following protracted abstinence in mice
TL;DR: While some brain regions were implicated in both drug and food seeking, other specific parts of the brain were activated for either sucrose or morphine relapse, which supports existing literature regarding relapse‐like behaviour in rats.
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Role of Dopamine 2 Receptor in Impaired Drug-Cue Extinction in Adolescent Rats
Isabel Zbukvic,Despina E. Ganella,Christina J. Perry,Heather B Madsen,Christopher R. Bye,Andrew J Lawrence,Jee Hyun Kim +6 more
TL;DR: It is observed that cue extinction reduced cue-induced reinstatement in adult but not adolescent rats, suggest that adolescents may be more susceptible to relapse due to a deficit in cue extinction learning, and highlight the significance of D2R signaling in the IL for cue extinction during adolescence.