H
Heiko Hiltscher
Researcher at University of Düsseldorf
Publications - 3
Citations - 195
Heiko Hiltscher is an academic researcher from University of Düsseldorf. The author has contributed to research in topics: Gene & Mutant. The author has an hindex of 3, co-authored 3 publications receiving 182 citations.
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Journal ArticleDOI
The redox-sensitive transcription factor Rap2.4a controls nuclear expression of 2-Cys peroxiredoxin A and other chloroplast antioxidant enzymes
Jehad Shaikhali,Isabelle Heiber,Thorsten Seidel,Elke Ströher,Heiko Hiltscher,Stefan Birkmann,Karl-Josef Dietz,Margarete Baier +7 more
TL;DR: The redox sensitivity of Rap2.4a establishes an efficient switch mechanism for redox control of nuclear gene activity of chloroplast antioxidants, in which Rap 2.4 is a redox-sensor and a transducer of redox information.
Journal ArticleDOI
The radical induced cell death protein 1 (RCD1) supports transcriptional activation of genes for chloroplast antioxidant enzymes.
Heiko Hiltscher,Radoslaw Rudnik,Jehad Shaikhali,Jehad Shaikhali,Isabelle Heiber,Marina Mellenthin,Iuri Meirelles Duarte,Günter Schuster,Uwe Kahmann,Margarete Baier,Margarete Baier,Margarete Baier +11 more
TL;DR: It is concluded that RCD1 protects plant cells from running into reactive oxygen species (ROS)-triggered programs, such as cell death and activation of pathogen-responsive genes (PR genes) and extra-plastidic antioxidant enzymes, by supporting the induction of the chloroplast antioxidant system.
Journal ArticleDOI
The Plant Immunity Regulating F-Box Protein CPR1 Supports Plastid Function in Absence of Pathogens.
Christiane Hedtmann,Wei Guo,Elena Reifschneider,Isabelle Heiber,Heiko Hiltscher,Joern van Buer,Aiko Barsch,Karsten Niehaus,Beth A. Rowan,Tobias Lortzing,Anke Steppuhn,Margarete Baier +11 more
TL;DR: expression studies in pathogen signaling mutants demonstrated that CPR1-mediated activation of genes for photosynthesis and chloroplast antioxidant protection is, in contrast to activation of pathogen responses, regulated independently from PAD4-controlled salicylic acid (SA) accumulation.