H
Heiner Mönig
Researcher at University of Kiel
Publications - 46
Citations - 876
Heiner Mönig is an academic researcher from University of Kiel. The author has contributed to research in topics: Cirrhosis & Pancreatitis. The author has an hindex of 17, co-authored 46 publications receiving 833 citations.
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Journal ArticleDOI
Longitudinal imaging reveals pituitary enlargement preceding hypoplasia in two brothers with combined pituitary hormone deficiency attributable to PROP1 mutation.
Felix G. Riepe,Carl-Joachim Partsch,Oliver Blankenstein,Heiner Mönig,Roland Pfäffle,Wolfgang G. Sippell +5 more
TL;DR: It is suggested that early pituitary enlargement may be the typical course in patients with PROP-1 mutations in whom pituitaries are not indicated, and altered expression of early transcription factors could be involved.
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Expression of leptin and leptin receptor during the development of liver fibrosis and cirrhosis.
TL;DR: Leptin is expressed at increasing levels in activated hepatic stellate cells in vivo, which may be a source of increased leptin tissue and serum levels contributing to the pathophysiology and morphological changes of chronic liver disease.
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Can peripheral blood γδ T cells predict osteonecrosis of the jaw? An immunological perspective on the adverse drug effects of aminobisphosphonate therapy
TL;DR: It is proposed Vγ9Vδ2 T cells show a strong potential to serve as harbingers of possible adverse immune effects of n‐BP therapy, particularly in those patients already having a compromised immune system as they may be most vulnerable to the development of conditions such as BAONJ.
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Selective inhibition of drug oxidation after simultaneous administration of two probe drugs, antipyrine and tolbutamide.
TL;DR: The two main inferences are that tolbutamide and antipyrine are metabolished by different forms of cytochrome P-450, and that a battery of model substrates is needed to investigate the inhibitory effects of a drug in man.
Journal ArticleDOI
Familial influences and obesity-associated metabolic risk factors contribute to the variation in resting energy expenditure: the Kiel Obesity Prevention Study
Anja Bosy-Westphal,Andreas Wolf,Frederike Bührens,Britta Hitze,Norbert Czech,Heiner Mönig,Oliver Selberg,Uta Settler,Maria Pfeuffer,Jürgen Schrezenmeir,Michael Krawczak,Manfred J. Müller +11 more
TL;DR: Obesity-related cardiometabolic risk factors contribute to interindividual variation in REE, with hypertension and insulin resistance being associated with a higher REE.