H
Henri R. Ford
Researcher at University of Southern California
Publications - 244
Citations - 10241
Henri R. Ford is an academic researcher from University of Southern California. The author has contributed to research in topics: Necrotizing enterocolitis & Pediatric trauma. The author has an hindex of 57, co-authored 238 publications receiving 9343 citations. Previous affiliations of Henri R. Ford include Children's Hospital Los Angeles & University of Pittsburgh.
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Journal ArticleDOI
Enterocyte TLR4 Mediates Phagocytosis and Translocation of Bacteria Across the Intestinal Barrier
Matthew D. Neal,Cynthia L. Leaphart,Ryan M. Levy,Jose M. Prince,Timothy R. Billiar,Simon C. Watkins,Jun Li,Selma Cetin,Henri R. Ford,Alan D. Schreiber,David J. Hackam +10 more
TL;DR: A novel mechanism by which bacterial translocation occurs is suggested and a critical role for TLR4 in the phagocytosis of bacteria by enterocytes in this process is suggested.
Journal ArticleDOI
Impact of pediatric trauma centers on mortality in a statewide system.
Douglas A. Potoka,Laura C. Schall,Mary J. Gardner,Perry W. Stafford,Andrew B. Peitzman,Henri R. Ford +5 more
TL;DR: Children treated at PTC or ATC AQ have significantly better outcome compared with those treated at ATC, and this difference in outcome may be attributable to the approach to operative and nonoperative management of head, liver, and spleen injuries at P TC.
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The human milk oligosaccharide disialyllacto-N-tetraose prevents necrotising enterocolitis in neonatal rats
Evelyn Jantscher-Krenn,Monica Zherebtsov,Caroline Nissan,Kerstin Goth,Yigit S. Guner,Natasha Naidu,Biswa Choudhury,Anatoly Grishin,Henri R. Ford,Lars Bode +9 more
TL;DR: HMO reduce NEC in neonatal rats and the effects are highly structure specific, and if these results translate to NEC in humans, DSLNT could be used to prevent or treat NEC in formula-fed infants, and its concentration in the mother's milk could serve as a biomarker to identify breast- fed infants at risk of developing this disorder.
Journal ArticleDOI
The role of inflammatory cytokines and nitric oxide in the pathogenesis of necrotizing enterocolitis
Henri R. Ford,Henri R. Ford,Simon C. Watkins,Simon C. Watkins,Kimberly K. Reblock,Kimberly K. Reblock,Marc I. Rowe,Marc I. Rowe +7 more
TL;DR: NO is produced in large quantity byEnterocytes in the intestinal wall of infants with NEC and leads to apoptosis of enterocytes in apical villi through peroxynitrite formation, and the levels of inflammatory cytokines and NO in the intestine of infants undergoing surgical resection for NEC are examined.
Journal ArticleDOI
Understanding the Susceptibility of the Premature Infant to Necrotizing Enterocolitis (NEC)
TL;DR: It is suggested that, although numerous bacterial and viral pathogens have been associated with NEC, no individual organism is known to be responsible for disease.