H
Henry G. Burger
Researcher at Prince Henry's Institute of Medical Research
Publications - 345
Citations - 22459
Henry G. Burger is an academic researcher from Prince Henry's Institute of Medical Research. The author has contributed to research in topics: Menopause & Follicle-stimulating hormone. The author has an hindex of 78, co-authored 345 publications receiving 21590 citations. Previous affiliations of Henry G. Burger include Walter and Eliza Hall Institute of Medical Research & Royal Children's Hospital.
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Prostatitis and Male Infertility: A Pilot Study. Possible Increase in Sperm Motility with Antibacterial Chemotherapy
TL;DR: It is concluded that antibacterial therapy may improve fertility in patients with low sperm motility, but more investigation is necessary before the role of genital tract infection in male infertility can be placed in its proper perspective.
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Serum prolactin levels and the value of bromocriptine in the treatment of anovulatory infertility.
TL;DR: Ovarian responses short of defined criteria for ovulation were induced initially in eight patients, but these progressed to full ovulatory responses in five patients, either on the same or increased doses of bromocriptine.
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Follicle-stimulating hormone induces dose-dependant stimulation of immunoreactive inhibin secretion during the follicular phase of the human menstrual cycle.
Jennifer Hee,J. MacNaughton,Mohan Bangah,M Zissimos,Philip I. McCloud,David L. Healy,Henry G. Burger +6 more
TL;DR: It was concluded that FSH stimulates INH in the follicular phase of the normal menstrual cycle, consistent with a physiological role for FSH in the regulation of granulosa cell production of inhibin.
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Chronic inhibitory effect of follicle-stimulating hormone (FSH)-suppressing protein (FSP) or follistatin on activin- and gonadotropin-releasing hormone-stimulated FSH synthesis and secretion in cultured rat anterior pituitary cells.
TL;DR: FSP inhibits not only the secretion but also the synthesis of FSH induced by activin and GnRH in long-term culture, and FSP may cause its inhibitory effects on GnRH by suppression of the protein kinase C system, and possibly by reduction of GnRH binding sites.
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The heart of the WHI study: time for hormone therapy policies to be revised
Amos Pines,David W. Sturdee,Alastair H. MacLennan,Hermann P.G. Schneider,Henry G. Burger,Anna Fenton +5 more
TL;DR: In the early postmenopausal period, coronary artery disease is no threat to hormone users and the WHI trial was stopped prematurely because an unvalidated global index, a measure balancing some of the risks and benefits of hormone use (quality of life was not included), exceeded a predetermined safety margin.